Brief Articles
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World J Gastroenterol. Jan 14, 2009; 15(2): 211-218
Published online Jan 14, 2009. doi: 10.3748/wjg.15.211
Role of bacterial and genetic factors in gastric cancer in Costa Rica
Sergio A Con, Hiroaki Takeuchi, Gil R Con-Chin, Vicky G Con-Chin, Nobufumi Yasuda, Reinaldo Con-Wong
Sergio A Con, Gil R Con-Chin, Vicky G Con-Chin, Reinaldo Con-Wong, Centro Digestivo Doctores Con-Mediplaza, Pavas 245-1200, San José, Costa Rica
Sergio A Con, Hiroaki Takeuchi, Department of Clinical Laboratory Medicine, Kochi Medical School, Kochi University, Nankoku-city, Kochi 783-8505, Japan
Nobufumi Yasuda, Department of Public Health, Kochi Medical School, Kochi University, Nankoku-city, Kochi 783-8505, Japan
Author contributions: Con SA designed and performed the research and wrote the paper; Takeuchi H, Con-Chin GR, Con-Chin VG and Con-Wong R contributed reagents/analytic tools; Yasuda N analyzed the data; Takeuchi H and Con-Wong R revised the paper.
Correspondence to: Dr. Sergio A Con, Centro Digestivo Doctores Con-Mediplaza, Pavas 245-1200, San José, Costa Rica. scon@gastrocolon.com
Telephone: +506-2201-7028
Fax: +506-2201-7033
Received: August 1, 2008
Revised: October 22, 2008
Accepted: October 29, 2008
Published online: January 14, 2009
Abstract

AIM: To evaluate several risk factors for gastric cancer (GC) in Costa Rican regions with contrasting GC incidence rate (GCIR).

METHODS: According to GCIR, 191 Helicobacter pylori (H pylori)-positive patients were classified into groups A (high GCIR, n = 101) and B (low GCIR, n = 90). Human DNA obtained from biopsy specimens was used in the determination of polymorphisms of the genes coding for interleukin (IL)-1&bgr; and IL-10 by PCR-RFLP, and IL-1RN by PCR. H pylori DNA extractions obtained from clinical isolates of 83 patients were used for PCR-based genotyping of H pylori cagA, vacA and babA2. Human DNA from gastric biopsies of 52 GC patients was utilized for comparative purposes.

RESULTS: Cytokine polymorphisms showed no association with GCIR variability. However, gastric atrophy, intestinal metaplasia and strains with different vacA genotypes in the same stomach (mixed strain infection) were more frequently found in group A than in group B, and cagA and vacA s1b were significantly associated with high GCIR (P = 0.026 and 0.041, respectively). IL-1&bgr;+3954_T/C (OR 2.1, 1.0-4.3), IL-1RN*2/L (OR 3.5, 1.7-7.3) and IL-10-592_C/A (OR 3.2, 1.5-6.8) were individually associated with GC, and a combination of these cytokine polymorphisms with H pylori vacA s1b and m1 further increased the risk (OR 7.2, 1.4-36.4).

CONCLUSION: Although a proinflammatory cytokine genetic profile showed an increased risk for developing GC, the characteristics of H pylori infection, in particular the status of cagA and vacA genotype distribution seemed to play a major role in GCIR variability in Costa Rica.

Keywords: Costa Rica; Gastric cancer; Helicobacter pylori; Host genetic factors