Basic Research
Copyright ©2008 The WJG Press and Baishideng. All rights reserved.
World J Gastroenterol. Dec 7, 2008; 14(45): 6936-6942
Published online Dec 7, 2008. doi: 10.3748/wjg.14.6936
Effects of Wy14643 on hepatic ischemia reperfusion injury in rats
Si-Qi Xu, Yuan-Hai Li, Sheng-Hong Hu, Ke Chen, Liu-Yi Dong
Si-Qi Xu, Yuan-Hai Li, Sheng-Hong Hu, Ke Chen, Department of Anesthesiology, First Affiliated Hospital of Anhui Medical University, Hefei 230022, Anhui Province, China
Liu-Yi Dong, Department of Pharmacology, Anhui Medical University, Hefei 230032, Anhui Province, China
Author contributions: Xu SQ, Li YH, Hu SH, Chen K and Dong LY designed the research; Xu SQ and Hu SH performed the research; Xu SQ, Li YH and Dong LY analyzed data; and Xu SQ and Li YH wrote the paper.
Correspondence to: Yuan-Hai Li, Professor, PhD, Department of Anesthesiology, First Affiliated Hospital of Anhui Medical University, Hefei 230022, Anhui Province, China. liyuanhai-1@163.com
Telephone: +86-551-2922057 Fax: +86-551-2922057
Received: July 29, 2008
Revised: September 9, 2008
Accepted: September 16, 2008
Published online: December 7, 2008
Abstract

AIM: To investigate the effects and possible mechanisms of Wy14643 on hepatic ischemia-reperfusion (I/R) injury in rats.

METHODS: Thirty male Sprague-Dawley rats weighing 220-280 g were randomly divided into five experimental groups: sham group (G1, n = 6): a sham operation was performed (except for liver I/R); I/R-untreated group (G2, n = 6): rats underwent liver ischemia for 90 min followed by reperfusion for 4 h; and I/R + Wy14643 groups (G3, G4, G5; n = 6): after the same surgical procedure as in group 2, animals were pretreated with Wy14643 at the dose of 1, 5 and 10 mg/kg 1 h before ischemia, respectively. Hepatic ischemia-reperfusion (I/R) was induced by clamping blood supply to the left lateral and median lobes of the liver for 90 min, and atraumatic clamp was removed for 4 h reperfusion. Blood samples and liver tissues were obtained at the end of reperfusion to assess serum and hepatic tissue homogenate aminotransferase (ALT), aspartate aminotransferase (AST), myeloperoxidase (MPO), serum interleukin-1β (IL-1β) and tumor necrosis factor alpha (TNF-α), as well as activity of superoxide dismutase (SOD) and content of malondialdehyde (MDA) in the hepatic tissue homogenate.

RESULTS: Hepatic I/R induced a significant increase in the serum levels of ALT, AST, TNF-α, IL-1β and MPO, as well as the levels of ALT, AST and MDA in the liver tissue homogenate, which were reduced by pretreatment with Wy14643 at the dose of 1, 5 and 10 mg/kg, respectively. The activity of SOD in the liver tissue homogenate was decreased after hepatic I/R, which was enhanced by Wy14643 pretreatment. In addition, serum and liver tissue homogenate ALT and AST in the Wy14643 10 mg/kg group were lower than in the Wy14643 1 mg/kg and 5 mg/kg groups, respectively.

CONCLUSION: Wy14643 pretreatment exerts significant protection against hepatic I/R injury in rats. The protective effects are possibly associated with enhancement of anti-oxidant and inhibition inflammation response.

Keywords: Wy14643; Liver; Ischemia-reperfusion; Effects