Case Report
Copyright ©2008 The WJG Press and Baishideng. All rights reserved.
World J Gastroenterol. Nov 14, 2008; 14(42): 6569-6571
Published online Nov 14, 2008. doi: 10.3748/wjg.14.6569
Isolated intestinal neurofibromatous proliferations in the absence of associated systemic syndromes
J Elliot Carter, Javier A Laurini
J Elliot Carter, Javier A Laurini, Department of Pathology, University of South Alabama, Alabama 36617-2293, United States
Author contributions: Carter JE and Laurini JA contributed equally to this work, and Carter JE and Laurini JA wrote the paper.
Correspondence to: J Elliot Carter, MD, Department of Pathology, University of South Alabama, 2451 Fillingim Street, Mobile, Alabama 36617-2293, United States. ecarter@usouthal.edu
Telephone: +1-251-4717790 Fax: +1-251-4717884
Received: May 8, 2008
Revised: June 1, 2008
Accepted: June 8, 2008
Published online: November 14, 2008
Abstract

Gastrointestinal tract involvement by neurofibromatous lesions is rare and occurs most frequently as one of the systemic manifestations of generalized neurofibromatosis type 1 (NF1). In this setting, the lesions may manifest as focal scattered neurofibromas or as an extensive diffuse neural hyperplasia designated ganglioneuromatosis. Occasionally, such lesions may be the initial sign of NF1 in patients without any other clinical manifestations of the disease. Rarely, cases of isolated neurofibromatosis of the large bowel with no prior or subsequent evidence of generalized neurofibromatosis have been documented. We present the case of a 52 year-old female with abdominal pain and alternating bowel habits. Colonoscopic evaluation revealed multiple small polyps in the cecum and the presence of nodular mucosa in the colon and rectum. Pathologic evaluation of the biopsies from the cecum, descending colon, sigmoid colon, and rectum revealed tangled fascicles of spindle cells expanding the lamina propia leading to separation of the intestinal crypts. Immunohistochemical stains helped confirm the diagnosis of diffuse intestinal neurofibromatosis. A thorough clinical evaluation failed to reveal any stigmata of generalized neurofibromatosis. This case represents a rare presentation of isolated intestinal neurofibromatosis in a patient without classic systemic manifestations of generalized neurofibromatosis and highlights the need in such cases for close clinical follow-up to exclude neurofibromatosis type I or multiple endocrine neoplasia type II.

Keywords: Neurofibromatous; Proliferations; Isolated; Intestinal