Case Report
Copyright ©2008 The WJG Press and Baishideng. All rights reserved.
World J Gastroenterol. Jul 21, 2008; 14(27): 4416-4419
Published online Jul 21, 2008. doi: 10.3748/wjg.14.4416
Rapid re-emergence of YMDD mutation of hepatitis B virus with hepatic decompensation after lamivudine retreatment
So Young Kwon, Won Hyeok Choe, Chang Hong Lee, Jong Eun Yeon, Kwan Soo Byun
So Young Kwon, Won Hyeok Choe, Chang Hong Lee, Department of Internal Medicine, Konkuk University, School of Medicine, Seoul 143-729, South Korea
Jong Eun Yeon, Kwan Soo Byun, Department of Internal Medicine, Korea University College of Medicine, Seoul 152-703, South Korea
Author contributions: Kwon SY wrote the paper; Choe WH and Lee CH reviewed the paper; Yeon JE and Byun KS provided the clinical advice.
Correspondence to: So Young Kwon, MD, Department of Internal Medicine, Konkuk University Hospital, 4-12 Hwayang-dong, Gwangjin-gu, Seoul 143-729, South Korea. sykwonmd@hotmail.com
Telephone: +82-2-20305010
Fax: +82-2-20307748
Received: April 13, 2008
Revised: May 26, 2008
Accepted: June 2, 2008
Published online: July 21, 2008
Abstract

Lamivudine has a high rate of antiviral resistance. Sequential treatment of anti-hepatitis B virus (HBV) is commonly used for lamivudine resistance. We report 4 cases of patients with rapid redetection of HBV mutants during the lamivudine retreatment. The four patients received lamivudine as an initial treatment of HBV and adefovir and lamivudine as a rescue therapy consecutively. HBV-DNA level, YMDD mutations and adefovir -resistant mutations (RFMP) were tested every 3 mo during the sequential treatment. All the patients showed YMDD mutations during the initial lamivudine therapy. After adefovir therapy for lamivudine resistance, they showed viral breakthrough. Adefovir was switched to lamivudine, however, it did not induce viral suppression at all, rather increased HBV-DNA with rapid reemergence of the YMDD mutations. All the patients had ALT flares, and hepatic decompensation occurred in two patients. After switching to adefovir combined with entecavir or lamivudine for a rescue therapy, the patients had reduction in HBV-DNA and ALT improvement. These cases demonstrated that lamivudine retreatment of patients with preexposed lamivudine resistance leads to rapid reemergence of YMDD mutation with significant viral rebounds and subsequent hepatic decompensation. Sequential administration of lamivudine in patients with a prior history of YMDD mutation should be abandoned.

Keywords: Hepatitis B; Lamivudine; Adefovir dipivoxil; Mutations