Rapid Communication
Copyright ©2008 The WJG Press and Baishideng. All rights reserved.
World J Gastroenterol. Jun 14, 2008; 14(22): 3518-3522
Published online Jun 14, 2008. doi: 10.3748/wjg.14.3518
Prevalence of Barrett’s esophagus in patients with moderate to severe erosive esophagitis
Nooman Gilani, Richard D Gerkin, Francisco C Ramirez, Shahina Hakim, Adam C Randolph
Nooman Gilani, Richard D Gerkin, Francisco C Ramirez, Shahina Hakim, Adam C Randolph, Department of Medicine and Research, Section of Gastroenterology, Carl. T. Hayden Veterans Administration Medical Center, Phoenix, Arizona 85012, United States
Author contributions: Gilani N designed research; Gilani N, Randolph AC and Hakim S performed research; Gilani N and Randolph AC analyzed data; Gerkin RD performed statistical analysis; Gilani N and Randolph AC wrote the paper; Gilani N, Ramirez FC, Randolph AC and Gerkin RD reviewed, edited and approved the final manuscript.
Correspondence to: Nooman Gilani, MD, FACG, FASGE, Chief of Endoscopy, Department of Gastroenterology (111G), Carl. T. Hayden VAMC, 650 E Indian School Road, Phoenix, Arizona 85012, United States. ngilani@pol.net
Telephone: +1-602-2775551
Fax: +1-602-2226562
Received: January 22, 2008
Revised: May 13, 2008
Accepted: May 20, 2008
Published online: June 14, 2008
Abstract

AIM: To investigate the proportion of patients with moderate-severe erosive esophagitis (EE) who will have Barrett’s esophagus (BE) after healing of inflammation.

METHODS: Patients with EE of Los Angeles (LA) class B, C and D who underwent follow-up endoscopy documenting complete mucosal healing.

RESULTS: A total of 86/169 patients were suspected of having BE (38 before healing and 48 after healing of EE) and, 46/86 eventually had the histological confirmation. At index esophago-gastro-duodenoscopy (EGD), BE was suspected in 38/169 (22%), and ultimately, histologically confirmed in 20 of these. In 11 patients where biopsies were performed in the presence of inflammation, BE was detected in 2 and missed in 5 (including 2 dysplasias). In 131/169 patients (77.5%), BE was not suspected at index EGD. After healing of EE though, 48 patients had suspicion of BE who underwent biopsies, and in 26 of these histology was positive for BE. The length of inflammation had a linear correlation with the length of BE (P = 0.01). Out of multiple variables to predict BE, only the suspicion at index endoscopy was statistically significant (P = 0.01).

CONCLUSION: BE was seen in 46/169 (27%) patients with EE of LA class B, C and D. The length of EE can predict the length of underlying BE segment. Even when suspected, BE and associated dysplasia can be missed in the presence of inflammation; therefore, repeat evaluation should be considered after complete healing of esophagitis.

Keywords: Erosive esophagitis; Barrett’s; Prevalence of Barrett’s; Gastroesophageal reflux