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World J Gastroenterol. Nov 28, 2007; 13(44): 5938-5943
Published online Nov 28, 2007. doi: 10.3748/wjg.v13.i44.5938
Comprehensive screening for reg1α gene rules out association with tropical calcific pancreatitis
Swapna Mahurkar, Seema Bhaskar, D Nageshwar Reddy, G Venkat Rao, Giriraj Ratan Chandak
Swapna Mahurkar, Seema Bhaskar, Giriraj Ratan Chandak, Genome Research Group, Centre for Cellular and Molecular Biology, Uppal Road, Hyderabad 500 007, India
D Nageshwar Reddy, G Venkat Rao, Asian Institute of Gastroenterology, Punjagutta, Hyderabad 500 082, India
Author contributions: All authors contributed equally to the work.
Supported by the Council of Scientific and Industrial Research, Ministry of Science and Technology, Government of India
Correspondence to: Giriraj Ratan Chandak, Centre for Cellular and Molecular Biology, Uppal Road, Hyderabad 500007, India. chandakgrc@ccmb.res.in
Telephone: +91-40-27160222-41 Fax: +91-40-27160591
Received: May 16, 2007
Revised: September 3, 2007
Accepted: October 26, 2007
Published online: November 28, 2007
Abstract

AIM: To investigate the allelic and haplotypic association of reg1α gene with tropical calcific pancreatitis (TCP). Since TCP is known to have a variable genetic basis, we investigated the interaction between mutations in the susceptibility genes, SPINK1 and CTSB with reg1α polymorphisms.

METHODS: We analyzed the polymorphisms in the reg1α gene by sequencing the gene including its promoter region in 195 TCP patients and 150 ethnically matched controls, compared their allele and haplotype frequencies, and their association with the pathogenesis and pancreaticolithiasis in TCP and fibro-calculous pancreatic diabetes.

RESULTS: We found 8 reported and 2 novel polymo-rphisms including an insertion-deletion polymorphism in the promoter region of reg1α. None of the 5’ UTR variants altered any known transcription factor binding sites, neither did any show a statistically significant association with TCP. No association with any reg1α variants was observed on dichotomization of patients based on their N34S SPINK1 or L26V CTSB status.

CONCLUSION: Polymorphisms in reg1α gene, including the regulatory variants singly or in combination with the known mutations in SPINK1 and/or CTSB genes, are not associated with tropical calcific pancreatitis.

Keywords: Tropical calcific pancreatitis; Lithostathine; Stone formation; Polymorphism; Haplotype