Editorial
Copyright ©2006 Baishideng Publishing Group Co., Limited. All rights reserved.
World J Gastroenterol. Feb 7, 2006; 12(5): 673-677
Published online Feb 7, 2006. doi: 10.3748/wjg.v12.i5.673
Novel mechanisms in functional dyspepsia
Rok Son Choung, Nicholas J Talley
Rok Son Choung, Nicholas J Talley, Clinical Enteric Neuroscience Translational and Epidemiological Research Program, Mayo Clinic, Rochester, MN, United States
Correspondence to: Nicholas J Talley, MD, PhD, Professor of Medicine, Mayo Clinic College of Medicine, Plummer 6-56, 200 First Street S.W., Rochester, MN 55905, United States. alley.nicholas@mayo.edu
Telephone: +1-507-266-1989 Fax: +1-507-538-7202
Received: August 5, 2005
Revised: August 28, 2005
Accepted: October 9, 2005
Published online: February 7, 2006
Abstract

Functional dyspepsia (FD) is a highly prevalent but heterogeneous disorder in which multiple pathogenetic mechanisms are involved. Although there are many studies that have investigated various pathophysiologic mechanisms, the underlying casual pathways associated with FD remain obscure. The currently proposed pathophysiologic mechanisms associated with FD include genetic susceptibility, delayed as well as accelerated gastric emptying, visceral hypersensitivity to acid or mechanical distention, impaired gastric accommodation, abnormal fundic phasic contractions, abnormal antro-duodenal motility, acute and chronic infections, and psychosocial comorbidity. A greater understanding of the abnormalities underlying FD may lead to improved management. The aim of this editorial is to provide a critical overview of current pathophysiologic concepts in functional dyspepsia.

Keywords: Functional dyspepsia; Gastric function; Pathophysiology