Clinical Research
Copyright ©2006 Baishideng Publishing Group Co., Limited. All rights reserved.
World J Gastroenterol. Sep 7, 2006; 12(33): 5336-5343
Published online Sep 7, 2006. doi: 10.3748/wjg.v12.i33.5336
Ursodeoxycholic acid improves gastrointestinal motility defects in gallstone patients
A Colecchia, G Mazzella, L Sandri, F Azzaroli, M Magliuolo, P Simoni, ML Bacchi-Reggiani, E Roda, D Festi
A Colecchia, G Mazzella, L Sandri, F Azzaroli, E Roda, D Festi, Department of Internal Medicine and Gastroenterology, University of Bologna, Bologna, Italy
M Magliuolo, P Simoni, Department of Pharmaceutical Sciences, University of Bologna, Bologna, Italy
ML Bacchi-Reggiani, Department of Cardiovascular Diseases, University of Bologna, Bologna, Italy
Correspondence to: Davide Festi, MD, Dipartmento di Medicina Interna e Gastroenterologia Policlinico S.Orsola-Malpighi Via Massarenti 9, Bologna 40138, Italy. festi@med.unibo.it
Telephone: +39-51-6364110 Fax: +39-51-6364123
Received: March 24, 2006
Revised: March 28, 2006
Accepted: May 22, 2006
Published online: September 7, 2006
Abstract

AIM: To simultaneously evaluate the presence of defects in gallbladder and gastric emptying, as well as in intestinal transit in gallstone patients (GS) and the effect of chronic ursodeoxycholic acid (UDCA) administration on these parameters and on serum bile acids and clinical outcome in GS and controls (CTR).

METHODS: After a standard liquid test meal, gallbla-dder and gastric emptying (by ultrasound), oroileal transit time (OITT) (by an immunoenzymatic technique) and serum bile acids (by HPLC) were evaluated before and after 3 mo of UDCA (12 mg/kg bw/d) or placebo administration in 10 symptomatic GS and 10 matched healthy CTR.

RESULTS: OITT was longer in GS than in CTR (P < 0.0001); UDCA significantly reduced OITT in GS (P < 0.0001), but not in CTR. GS had longer gastric half-emptying time (t1/2) than CTR (P < 0.0044) at baseline; after UDCA, t1/2 significantly decreased (P < 0.006) in GS but not in CTR. Placebo administration had no effect on gastric emptying and intestinal transit in both GS and CTR.

CONCLUSION: The gallstone patient has simultaneous multiple impairments of gallbladder and gastric emptying, as well as of intestinal transit. UDCA administration restores these defects in GS, without any effect in CTR. These results confirm the pathogenetic role of gastrointestinal motility in gallstone disease and suggest an additional mechanism of action for UDCA in reducing bile cholesterol supersaturation.

Keywords: Bile acids; Biliary cholesterol; Deoxycholic acid; Gallbladder emptying; Gastric emptying; Intestinal transit; Tauroursodeoxycholic acid