Brief Reports
Copyright ©2005 Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Feb 21, 2005; 11(7): 1032-1036
Published online Feb 21, 2005. doi: 10.3748/wjg.v11.i7.1032
Helicobacter pylori infection and low serum pepsinogen I level as risk factors for gastric carcinoma
Arto Kokkola, Johanna Louhimo, Pauli Puolakkainen, Henrik Alfthan, Caj Haglund, Hilpi Rautelin
Arto Kokkola, Johanna Louhimo, Pauli Puolakkainen, Caj Haglund, Department of Surgery, Helsinki University Central Hospital, Helsinki, Finland
Henrik Alfthan, Department of Clinical Chemistry, Helsinki University Central Hospital, Helsinki, Finland
Hilpi Rautelin, Department of Bacteriology and Immunology, Haartman Institute, University of Helsinki and HUSLAB, Helsinki University Central Hospital Laboratory, Helsinki, Finland
Author contributions: All authors contributed equally to the work.
Correspondence to: Arto Kokkola, M.D., Department of Surgery, Meilahti Hospital, PO Box 340 (Haartmaninkatu 4), FIN-00029 HUS, Finland. arto.kokkola@hus.fi
Telephone: +358-50-4271048 Fax: +358-9-47174675
Received: May 29, 2004
Revised: June 2, 2004
Accepted: July 27, 2004
Published online: February 21, 2005
Abstract

AIM: To study whether examination of CagA antibodies could increase the odds ratio for gastric cancer in a case-control study, and how often other serum markers of gastric cancer risk could be found in Helicobacter pylori -negative patients.

METHODS: H pylori CagA and parietal cell antibodies (PCAs), and serum pepsinogen I (SPGI) levels were compared between patients with gastric cancer and controls who received endoscopic examination due to reasons other than gastrointestinal malignancy.

RESULTS: The odds ratio (OR) for gastric cancer was 2.9 (95% CI 1.4-5.8) in H pylori+ patients, and 2.4 (95% CI 1.2-4.9) in CagA+ patients. When results of H pylori and CagA antibodies were combined, OR increased to 5.0 (95% CI 2.5-10.0). Furthermore, if cardia cancer patients were excluded, the OR increased to 6.8 (95% CI 3.1-14.8). Among patients with a low SPGI level, the OR was 12.0 (95% CI 4.1-35.3). However, the risk was significant only in the older age group. The number of patients with low SPGI was significantly higher in H pylori-/CagA+ patients as compared to other cancer patients.

CONCLUSION: Examination of both H pylori and CagA antibodies increases the OR for gastric cancer in our case-control study. CagA antibodies are important in detecting previous H pylori infection in advanced atrophic gastritis or cancer when spontaneous decline of H pylori antibodies occurs. SPGI may be helpful in screening elderly gastric cancer patients.

Keywords: Gastric cancer; Risk factor; Atrophic gastritis