Basic Research
Copyright ©The Author(s) 2004. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. May 1, 2004; 10(9): 1325-1328
Published online May 1, 2004. doi: 10.3748/wjg.v10.i9.1325
Modulation of Kupffer cells on hepatic drug metabolism
Hong Ding, Jing Tong, Shi-Cheng Wu, Deng-Ke Yin, Xian-Fen Yuan, Jian-Yuan Wu, Jun Chen, Gang-Gang Shi
Hong Ding, Gang-Gang Shi, Medical College, Shantou University, Shantou 515031, Guangdong Province, China
Jing Tong, Shi-Cheng Wu, Deng-Ke Yin, Xian-Fen Yuan, Jian-Yuan Wu, Jun Chen, Department of Pharmacology, College of Pharmacy, Wuhan University, Wuhan 430072, Hubei Province, China
Author contributions: All authors contributed equally to the work.
Supported by Postdoctor Science Foundation of China, No. 2002032238; the Major State Basic Research Deaelopment Program of China, No. 2002ccc00300
Correspondence to: Jing Tong, College of Pharmacy, Wuhan University, Wuhan 430072, Hubei Province, China. tongjing2002@etang.com
Telephone: +86-27-87682339 Fax: +86-27-87682339
Received: August 26, 2003
Revised: September 20, 2003
Accepted: October 7, 2003
Published online: May 1, 2004
Abstract

AIM: To observe the effects of Kupffer cells on hepatic drug metabolic enzymes.

METHODS: Kunming mice were ip injected with GdCl310, 20, 40 mg/kg to decrease the number and block the function of kupffer cells selectively. The contents of drug metabolic enzymes, cytochrome P450, NADPH-cytochrom C redutase (NADPH-C), aniline hydroxylase (ANH), aminopyrine N-demethylase (AMD), erythromycin N-demethylase (EMD), and glutathione s-transferase (mGST) in hepatic microsome and S9-GSTpi, S9-GST in supernatant of 9000 g were accessed 1 d after the injection. The time course of alteration of drug metabolic enzymes was observed on d 1, 3, and 6 treated with a single dose GdCl3. Mice were treated with Angelica sinensis polysaccharides (ASP) of 30, 60, 120 mg/kg, ig, qd × 6 d, respectively and the same assays were performed.

RESULTS: P450 content and NADPH-C, ANH, AMD, and EMD activities were obviously reduced 1 d after Kupffer cell blockade. However, mGST and S9-GST activities were significantly increased. But no relationship was observed between GdCl3 dosage and enzyme activities. With single dose GdCl3 treatment, P450 content, NADPH-C, and ANH activities were further decreased following Kupffer cell blockade lasted for 6 d, by 35.7%, 50.3%, 36.5% after 3 d, and 57.9%, 57.9%, 63.2% after 6 d, respectively. On the contrary, AMD, EMD, mGST, and S9-GST activities were raised by 36.5%, 71.9%, 23.1%, 35.7% after 3 d, and 155%, 182%, 21.5%, 33.7% after 6 d, respectively. Furthermore, the activities of drug metabolic enzymes were markedly increased after 30 mg/kg ASP treatment, and decreased significantly after 120 mg/kg ASP treatment. No change in activity of S9-GSTpi was observed in the present study.

CONCLUSION: Kupffer cells play an important role in the modulation of drug metabolic enzymes. The changes of drug metabolic enzyme activities depend on the time of kupffer cell blockade and on the degree of Kupffer cells activated. A low concentration of ASP increases the activities of drug metabolic enzymes, but a high concentration of ASP decreases the activities of drug metabolic enzymes.

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