Brief Reports
Copyright ©The Author(s) 2004. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Sep 1, 2004; 10(17): 2557-2559
Published online Sep 1, 2004. doi: 10.3748/wjg.v10.i17.2557
Helicobacter pylori in gastric corpus of patients 20 years after partial gastric resection
Christian Kirsch, Ahmed Madisch, Petja Piehler, Ekkehard Bayerdörffer, Manfred Stolte, Stephan Miehlke
Christian Kirsch, Ahmed Madisch, Stephan Miehlke, Medical Department I, Technical University Hospital, Dresden, Germany
Petja Piehler, Community Hospital, Kitzbühl, Germany
Ekkehard Bayerdörffer, Department of Internal Medicine, University Hospital, Marburg, Germany
Manfred Stolte, Institute of Pathology, Klinikum Bayreuth, Germany
Author contributions: All authors contributed equally to the work.
Correspondence to: Dr. Stephan Miehlke, Medical Department I, Technical University Hospital Carl Gustav Carus, Fetscherstraβe 74, D-01307 Dresden, Germany. miehlke@mk1.med.tu-dresden.de
Telephone: +49-351-4585645 Fax: +49-351-4584394
Received: March 23, 2004
Revised: April 4, 2004
Accepted: April 29, 2004
Published online: September 1, 2004
Abstract

AIM: To determine the long-term prevalence of Helicobacter pylori (H pylori) gastritis in patients after partial gastric resection due to peptic ulcer, and to compare the severity of H pylori-positive gastritis in the corpus mucosa between partial gastrectomy patients and matched controls.

METHODS: Endoscopic biopsies were obtained from 57 patients after partial gastric resection for histological examination using hematoxylin/eosin and Warthin-Starry staining. Gastritis was graded according to the updated Sydney system. Severity of corpus gastritis was compared between H pylori-positive partial gastrectomy patients and H pylori-positive duodenal ulcer patients matched for age and gender.

RESULTS: In partial gastrectomy patients, surgery was performed 20 years (median) prior to evaluation. In 25 patients (43.8%) H pylori was detected histologically in the gastric remnant. Gastric atrophy was more common in H pylori-positive compared to H pylori-negative partial gastrectomy patients (P < 0.05). The severity of corpus gastritis was significantly lower in H pylori-positive partial gastrectomy patients compared to duodenal ulcer patients (P < 0.01). There were no significant differences in the activity of gastritis, atrophy and intestinal metaplasia between the two groups.

CONCLUSION: The long-term prevalence of H pylori gastritis in the gastric corpus of patients who underwent partial gastric resection due to peptic ulcer disease is comparable to the general population. The expression of H pylori gastritis in the gastric remnant does not resemble the gastric cancer phenotype.

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