Published online Aug 15, 2004. doi: 10.3748/wjg.v10.i16.2330
Revised: February 2, 2004
Accepted: February 21, 2004
Published online: August 15, 2004
AIM: To investigate the effect of interleukin-12 p40 gene (IL12B) 3’-untranslated region polymorphism on the outcome of HCV infection.
METHODS: A total of 133 patients who had been infected with HCV for 12-25 (18.2 ± 3.8) years, were enrolled in this study. Liver biochemical tests were performed with an automated analyzer and HCV RNA was detected by fluorogenic quantitative polymerase chain reaction. B-mode ultrasound was used for liver examination. Polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP) was used for the detection of IL12B (1188A/C) polymorphism.
RESULTS: Self-limited infection was associated with AC genotype (OR = 3.48; P = 0.001) and persistent infection was associated with AA genotype (OR = 0.34; P = 0.014) at site 1188 of IL12B. In patients with persistent HCV infection, no significant differences were found regarding the age, gender, duration of infection and biochemical characteristics (P > 0.05). According to B-mode ultrasound imaging and clinical diagnosis, patients with persistent infection were divided into groups based on the severity of infection. No significant differences were found in the frequency of IL-12 genotype (1188A/C) between different groups (P > 0.05).
CONCLUSION: The polymorphism of IL12B (1188A/C) appears to have some influence on the outcome of HCV infection.