Brief Reports
Copyright ©The Author(s) 2004. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Jun 15, 2004; 10(12): 1830-1833
Published online Jun 15, 2004. doi: 10.3748/wjg.v10.i12.1830
Effects of tegaserod on Fos, substance P and calcitonin gene-related peptide expression induced by colon inflammation in lumbarsacral spinal cord
Yi-Ning Sun, Jin-Yan Luo
Yi-Ning Sun, Jin-Yan Luo, Department of Gastroenterology, Second Hospital of Xi’an Jiaotong University, Xi’an 710004, Shaanxi Province, China
Author contributions: All authors contributed equally to the work.
Correspondence to: Professor Jin-Yan Luo, Department of Gastroenterology, Second Hospital of Xi’an Jiaotong University, Xi’an 710004, Shaanxi Province, China
Telephone: +86-29-87678758
Received: September 6, 2003
Revised: October 15, 2003
Accepted: October 22, 2003
Published online: June 15, 2004
Abstract

AIM: To investigate the mechanisms of tegaserod, a partial 5-HT4 agonist, in reducing visceral sensitivity by observing Fos, substance P (SP) and calcitonin gene-related peptide (CGRP) expression in the lumbarsacral spinal cord induced by colonic inflammation in rats.

METHODS: Twenty-four male rats with colonic inflammation induced by intraluminal instillation of trinitrobenzenesulfonic acid (TNBS) were divided into 3 groups. Treatment group 1: intra-gastric administration of tegaserod, 2 mg/kg·d; Treatment group 2: intra-gastric administration of tegaserod, 1 mg/kg·d; Control group: intra-gastric administration of saline, 2.0 mL/d. After 7 d of intra-gastric administration, lumbarsacral spinal cord was removed and processed for Fos, SP and CGRP immunohistochemistry.

RESULTS: In rats of the control group, the majority of Fos labeled neurons was localized in deeper laminae of the lumbarsacral spinal cord (L5-S1). SP and CGRP were primarily expressed in the superficial laminae of the spinal cord after TNBS injection. Intra-gastric administration of tegaserod (2 mg/kg·d) resulted in a significant decrease of Fos labeled neurons (22.0 ± 7.7) and SP density (12.5 ± 1.4) in the dorsal horn in the lumbarsacral spinal cord compared to those of the control group (62.2 ± 18.9, 35.9 ± 8.9, P < 0.05). However, CGRP content in dorsal horn did not significantly reduce in rats of treatment group 1 (1.2 ± 1.1) compared to that of the control group (2.8 ± 2.4, P > 0.05). Neither Fos expression nor SP or CGRP density in the dorsal horn significantly declined in rats of treatment group 2 compared to those of the control group (P > 0.05).

CONCLUSION: Tegaserod can significantly reduce Fos labeled neurons in the lumbarsacral spinal cord induced by colonic inflammation. Tegaserod may reduce visceral sensitivity by inhibiting SP expression in the dorsal horn of spinal cord.

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