Epigenetics and DNA methylation in cancer

doi:10.5528/wjtm.v4.i1.11

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Apr 12, 2015 (publication date) through Nov 4, 2024

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World Journal of Translational Medicine

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2220-6132

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Transl Med. Apr 12, 2015; 4(1): 11-24
Published online Apr 12, 2015. doi: 10.5528/wjtm.v4.i1.11

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Figure 8 Epi-miRNA functions in cancer cells. Epi-miRNAs (in red) directly target epigenetic effectors (black boxes) and indirectly affect the expression of epigenetically regulated miRNAs and protein coding genes (white boxes), contributing to carcinogenesis. TSGs: Tumor suppressor genes; DNMT: DNA methyltransferase; HDAC: Histone deacetylase; EZH2: Enhancer of zeste homolog 2; BCL6: B-cell CLL/lymphoma 6; CDK6: Cyclin-dependent kinase 6; SP1: Sp1 transcription factor; RBL2: Retinoblastoma-like 2 (p130); CH₃: Methyl group. Adapted by Fabbri et al[25], 2013.

Citation: Lattanzio L, Lo Nigro C. Epigenetics and DNA methylation in cancer. World J Transl Med 2015; 4(1): 11-24
URL: https://www.wjgnet.com/2220-6132/full/v4/i1/11.htm
DOI: https://dx.doi.org/10.5528/wjtm.v4.i1.11