Hepatic encephalopathy as a complication of liver disease

doi:10.3748/wjg.v7.i2.152

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Apr 15, 2001 (publication date) through Nov 29, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Apr 15, 2001; 7(2): 152-156
Published online Apr 15, 2001. doi: 10.3748/wjg.v7.i2.152

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Figure 1 Parietal ¹H-MNR spectra from (A) a healthy person, patients with posthepatitic cirrhosis and latent (subclinical) HE (B) and manifest grade I-II HE (C). An increase in the glutamine/ glutamate signal (Glx) and a decrease of the inositol signal (Ino) is observed. Further abbreviations: Cho, choline; Cr, creatine; N-acetylaspartate (NAA)[18].

Citation: Dahl SV, Kircheis G, Haussinger D. Hepatic encephalopathy as a complication of liver disease. World J Gastroenterol 2001; 7(2): 152-156
URL: https://www.wjgnet.com/1007-9327/full/v7/i2/152.htm
DOI: https://dx.doi.org/10.3748/wjg.v7.i2.152