Mechanisms of regulation of PFKFB expression in pancreatic and gastric cancer cells

doi:10.3748/wjg.v20.i38.13705

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Oct 14, 2014 (publication date) through Dec 22, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Oct 14, 2014; 20(38): 13705-13717
Published online Oct 14, 2014. doi: 10.3748/wjg.v20.i38.13705

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Figure 7 Representative polyacrylamide gel autoradiograph employed in a typical ribonuclease protection assay of different 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase genes (PFKFB-1, PFKFB-2, PFKFB-3, and PFKFB-4), GLUT1, hypoxia inducible factor-1α, and different alternative splice variants of VEGF-A in gastric malignant tumors (T) and non-malignant tissue counterparts (N) from same patients. The 18S rRNA expressions were used as control of RNA quantity used for analysis[32]. HIF: Hypoxia inducible factor; PFKFB: 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase.

Citation: Minchenko OH, Tsuchihara K, Minchenko DO, Bikfalvi A, Esumi H. Mechanisms of regulation of PFKFB expression in pancreatic and gastric cancer cells. World J Gastroenterol 2014; 20(38): 13705-13717
URL: https://www.wjgnet.com/1007-9327/full/v20/i38/13705.htm
DOI: https://dx.doi.org/10.3748/wjg.v20.i38.13705