Helicobacter pylori infection: New pathogenetic and clinical aspects

doi:10.3748/wjg.v20.i21.6386

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Jun 7, 2014 (publication date) through Feb 21, 2025

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jun 7, 2014; 20(21): 6386-6399
Published online Jun 7, 2014. doi: 10.3748/wjg.v20.i21.6386

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Figure 3 Targets of non-phosphorylated cytotoxin-associated gene a product. Based on the article from Current Opinion in Microbiology, Hatakeyama M, SagA of CagA in Helicobacter pylori pathogenesis, 11, 30-37, Copyright (2008), with permission from Elsevier[7]. CagA: Cytotoxin-associated gene A product; PLC: Phospholipase C gamma; PAR1: Kinase partitioning-defective 1b; ZO-1: Zonula occludens 1; JAM: Junctional adhesion molecule A; NFκB: Nuclear factor κB; TNF-α: Tumor necrosis factor-α; IL: Interleukin.

Citation: Hagymási K, Tulassay Z. Helicobacter pylori infection: New pathogenetic and clinical aspects. World J Gastroenterol 2014; 20(21): 6386-6399
URL: https://www.wjgnet.com/1007-9327/full/v20/i21/6386.htm
DOI: https://dx.doi.org/10.3748/wjg.v20.i21.6386