Roles of the PI3K/Akt pathway in Epstein-Barr virus-induced cancers and therapeutic implications

doi:10.5501/wjv.v1.i6.154

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Dec 12, 2012 (publication date) through Jan 31, 2025

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World Journal of Virology

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2220-3249

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Virol. Dec 12, 2012; 1(6): 154-161
Published online Dec 12, 2012. doi: 10.5501/wjv.v1.i6.154

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Figure 1 Epstein-Barr virus activates the phosphoinositide 3-kinase/protein kinase B pathway to transform cells. The Epstein-Barr virus latent proteins latent membrane protein (LMP)1 and LMP2A activate the phosphoinositide 3-kinase/protein kinase B (PI3K/Akt) pathway, which promotes carcinogenesis by increasing cell proliferation, genomic instability and cytoskeleton changes and by decreasing apoptosis.

Citation: Chen J. Roles of the PI3K/Akt pathway in Epstein-Barr virus-induced cancers and therapeutic implications. World J Virol 2012; 1(6): 154-161
URL: https://www.wjgnet.com/2220-3249/full/v1/i6/154.htm
DOI: https://dx.doi.org/10.5501/wjv.v1.i6.154