Secondary prevention of ischaemic stroke

doi:10.5316/wjn.v3.i4.97

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Dec 28, 2013 (publication date) through Jun 9, 2024

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World Journal of Neurology

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2218-6212

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Neurol. Dec 28, 2013; 3(4): 97-114
Published online Dec 28, 2013. doi: 10.5316/wjn.v3.i4.97

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Figure 1 Medical and surgical treatments which have been proved to be effective for secondary prevention after stroke or transient ischaemic attacks in the last decades. OAC: Oral anticoagulants; CEA: Carotid endarterectomy; AF: Atrial fibrillation.

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Figure 2 Old and new antiplatelet agents and their mechanism of action. PAR1: Proteinase activated receptor 1; ADP: Adenosine diphosphate; TXA₂: Thromboxane A₂; cAMP: Cyclic adenosine monophosphate; COX₁: Cyclooxygenase-1; 5-HT2A: 5-hydroxytryptamine (serotinin) receptor 2A.

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Figure 3 Vitamin K antagonist and new anticoagulant mechanism of action. F: Factor.

Citation: Volonghi I, Padovani A, Zotto ED, Giossi A, Costa P, Morotti A, Poli L, Pezzini A. Secondary prevention of ischaemic stroke. World J Neurol 2013; 3(4): 97-114
URL: https://www.wjgnet.com/2218-6212/full/v3/i4/97.htm
DOI: https://dx.doi.org/10.5316/wjn.v3.i4.97