Not all arrestins are created equal: Therapeutic implications of the functional diversity of the β-arrestins in the heart

doi:10.4330/wjc.v11.i2.47

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Feb 26, 2019 (publication date) through Nov 7, 2024

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World Journal of Cardiology

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1949-8462

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Cardiol. Feb 26, 2019; 11(2): 47-56
Published online Feb 26, 2019. doi: 10.4330/wjc.v11.i2.47

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Figure 1 The functional distinction between β-arrestin-1 and β-arrestin-2 in cardiac myocytes. ATP: Adenosine triphosphate; P: Phosphorylation; SR: Sarcoplasmic reticulum; SUMO1: Small ubiquitin-like modifier protein-1; PLN: Phospholamban; PIP₂: Phosphatidyl-inositol 4,5-bisphosphate; IP₃: Inositol 1,4,5-trisphosphate; DAG: 1,2-Diacylglycerol; EGFR: Epidermal growth factor receptor; AR: Adrenergic receptor.

Citation: Lymperopoulos A, Wertz SL, Pollard CM, Desimine VL, Maning J, McCrink KA. Not all arrestins are created equal: Therapeutic implications of the functional diversity of the β-arrestins in the heart. World J Cardiol 2019; 11(2): 47-56
URL: https://www.wjgnet.com/1949-8462/full/v11/i2/47.htm
DOI: https://dx.doi.org/10.4330/wjc.v11.i2.47