Prospective Study
Copyright ©The Author(s) 2017.
World J Hepatol. Jul 8, 2017; 9(19): 857-866
Published online Jul 8, 2017. doi: 10.4254/wjh.v9.i19.857
Figure 1
Figure 1 Flow-chart according to sample recruitment. ALT: Alanine transferase.
Figure 2
Figure 2 Path model for the effects of hepatic steatosis via systolic and diastolic blood pressure on left ventricular mass index in the whole study population (n = 1298). χ2 = 3.2, df = 3, P = 0.36; RMSEA < 0.01; CFI > 0.99. Indirect Effect via BPsys0: β = -0.20; s.e. = 0.10; P = 0.07; Indirect Effect via BPdia0: β = 0.03; s.e. = 0.06; P = 0.60. FLD: Fatty liver disease; LVMI: Left ventricular mass index; BPsys: Systolic blood pressure; BPdia: Diastolic blood pressure; RMSEA: Root mean square error; CFI: Comparative fit index; s.e.: Standard error.
Figure 3
Figure 3 Path model for the effects of hepatic steatosis via systolic and diastolic blood pressure on left ventricular mass index in the subgroup of individuals without medication (n = 811). χ2 = 1.9, df = 3, P = 0.60; RMSEA < 0.01; CFI > 0.99. Indirect Effect via BPsys0: β = -0.30; s.e. = 0.17; P = 0.07; Indirect Effect via BPdia0: β = 0.15; s.e. = 0.14; P = 0.30. FLD: Fatty liver disease; LVMI: Left ventricular mass index; BPsys: Systolic blood pressure; BPdia: Diastolic blood pressure; RMSEA: Root mean square error; CFI: Comparative fit index; s.e.: Standard error.