Oxidative stress modulation in hepatitis C virus infected cells

doi:10.4254/wjh.v7.i29.2880

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Dec 18, 2015 (publication date) through Nov 3, 2024

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World Journal of Hepatology

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1948-5182

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Hepatol. Dec 18, 2015; 7(29): 2880-2889
Published online Dec 18, 2015. doi: 10.4254/wjh.v7.i29.2880

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Figure 1 Possible interactions/mechanisms of antioxidant agents with reported anti-hepatitis C virus effect. SOD: Superoxide dismutase; SAM: S-adenosylmethionine; ASA: Acetylsalicylic acid; Zn: Zinc; IFN: Interferon.

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Figure 2 Cell signaling pathways modulated by increased reactive oxygen species levels in hepatitis C virus infected cells. ROS: Reactive oxygen species; HCV: Hepatitis C virus; GSH: Glutathione system; Trx: Thioredoxin; PI3K: Phosphoinositide-3 kinase.

Citation: Lozano-Sepulveda SA, Bryan-Marrugo OL, Cordova-Fletes C, Gutierrez-Ruiz MC, Rivas-Estilla AM. Oxidative stress modulation in hepatitis C virus infected cells. World J Hepatol 2015; 7(29): 2880-2889
URL: https://www.wjgnet.com/1948-5182/full/v7/i29/2880.htm
DOI: https://dx.doi.org/10.4254/wjh.v7.i29.2880