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For: Herbelet S, De Paepe B, De Bleecker JL. Abnormal NFAT5 Physiology in Duchenne Muscular Dystrophy Fibroblasts as a Putative Explanation for the Permanent Fibrosis Formation in Duchenne Muscular Dystrophy. Int J Mol Sci 2020;21:E7888. [PMID: 33114289 DOI: 10.3390/ijms21217888] [Cited by in Crossref: 5] [Cited by in F6Publishing: 5] [Article Influence: 2.5] [Reference Citation Analysis]
Number Citing Articles
1 Gong F, Li X, Zhang H, Wu J, Ma G, Zhang B, Gao J, Ding Y, Huang Y, Xia K, Cheng S, Zhou X, Shi J, Zhao F, Chen G. MiR-192-5p Alleviated Fibrosis and Inflammatory Responses of Tendon Cells by Targeting NFAT5. Computational and Mathematical Methods in Medicine 2022;2022:1-11. [DOI: 10.1155/2022/6481846] [Reference Citation Analysis]
2 Merckx C, Cosemans G, Zschüntzsch J, Raedt R, Schmidt J, De Paepe B, De Bleecker JL. Description of Osmolyte Pathways in Maturing Mdx Mice Reveals Altered Levels of Taurine and Sodium/Myo-Inositol Co-Transporters. Int J Mol Sci 2022;23:3251. [PMID: 35328671 DOI: 10.3390/ijms23063251] [Cited by in Crossref: 1] [Cited by in F6Publishing: 1] [Article Influence: 1.0] [Reference Citation Analysis]
3 Herbelet S, Merckx C, De Paepe B. The PKA-p38MAPK-NFAT5-Organic Osmolytes Pathway in Duchenne Muscular Dystrophy: From Essential Player in Osmotic Homeostasis, Inflammation and Skeletal Muscle Regeneration to Therapeutic Target. Biomedicines 2021;9:350. [PMID: 33808305 DOI: 10.3390/biomedicines9040350] [Cited by in F6Publishing: 2] [Reference Citation Analysis]
4 Peristeri E, Dardiotis E. Editorial for Special Issue "Genetic Basis and Epidemiology of Myopathies". Int J Mol Sci 2021;22:2152. [PMID: 33671495 DOI: 10.3390/ijms22042152] [Reference Citation Analysis]
5 Herbelet S, De Paepe B, De Bleecker JL. Description of a Novel Mechanism Possibly Explaining the Antiproliferative Properties of Glucocorticoids in Duchenne Muscular Dystrophy Fibroblasts Based on Glucocorticoid Receptor GR and NFAT5. Int J Mol Sci 2020;21:E9225. [PMID: 33287327 DOI: 10.3390/ijms21239225] [Cited by in Crossref: 2] [Cited by in F6Publishing: 3] [Article Influence: 1.0] [Reference Citation Analysis]