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Cited by in F6Publishing
For: Li Y, Yang S, Yang S. Verteporfin Inhibits the Progression of Spontaneous Osteosarcoma Caused by Trp53 and Rb1 Deficiency in Ctsk-Expressing Cells via Impeding Hippo Pathway. Cells 2022;11:1361. [DOI: 10.3390/cells11081361] [Cited by in Crossref: 6] [Cited by in F6Publishing: 6] [Article Influence: 6.0] [Reference Citation Analysis]
Number Citing Articles
1 Li J, Wang R, Liu Y, Wu Y, Han L, Zheng L, Bao Z. FKA-A NPs enhances PTX-A NPs efficacy to suppress ovarian cancer via regulating Skp2/YAP pathway. Fundam Clin Pharmacol 2023;37:125-36. [PMID: 36028983 DOI: 10.1111/fcp.12828] [Reference Citation Analysis]
2 Li Y, Yang S, Yang S. Rb1 negatively regulates bone formation and remodeling through inhibiting transcriptional regulation of YAP in Glut1 and OPG expression and glucose metabolism in male mice. Mol Metab 2022;66:101630. [PMID: 36343919 DOI: 10.1016/j.molmet.2022.101630] [Reference Citation Analysis]
3 Popova E, Tkachev S, Reshetov I, Timashev P, Ulasov I. Imaging Hallmarks of Sarcoma Progression Via X-ray Computed Tomography: Beholding the Flower of Evil. Cancers 2022;14:5112. [DOI: 10.3390/cancers14205112] [Reference Citation Analysis]
4 Li Y, Yang ST, Yang S. Trp53 controls chondrogenesis and endochondral ossification by negative regulation of TAZ activity and stability via β-TrCP-mediated ubiquitination. Cell Death Discov 2022;8:317. [PMID: 35831272 DOI: 10.1038/s41420-022-01105-2] [Reference Citation Analysis]
5 Li Y, Yang S, Liu Y, Yang S. Mice with Trp53 and Rb1 deficiency in chondrocytes spontaneously develop chondrosarcoma via overactivation of YAP signaling. Cell Death Dis 2022;13:570. [PMID: 35760773 DOI: 10.1038/s41419-022-04916-4] [Cited by in Crossref: 1] [Cited by in F6Publishing: 1] [Article Influence: 1.0] [Reference Citation Analysis]
6 Li Y, Yang S, Liu Y, Qin L, Yang S. IFT20 governs mesenchymal stem cell fate through positively regulating TGF-β-Smad2/3-Glut1 signaling mediated glucose metabolism. Redox Biol 2022;54:102373. [PMID: 35751983 DOI: 10.1016/j.redox.2022.102373] [Cited by in F6Publishing: 2] [Reference Citation Analysis]