Study on the classification of chronic gastritis at molecular biological level

Figure 1 Mouth of gastric glands, like craters, The concaver walls have round or oval epithelial cells, of almost same size. × 160.

Figure 2 Craters are deformed, the crater periphery projections are of different heights and width. × 320.

Figure 3 On cell surfaces, S-shape Helicobacter pylori are found. × 2500.

Figure 4 Micro ulcers. × 320.

Figure 5 Epithelial cells of crater concave walls are atrophic, denatured, of different sizes and deranged. × 640.

Figure 6 Inflammatory cell infiltration. × 2500.

Figure 7 Glands propria of grid framework structure. × 320.

Figure 8 Surface of IM gastric mucosa epithelial is thickly coated, villi are invisible, intercellular boundaries are not clear. × 320.

Figure 9 Normal epithelial nuclei are round, there are RERS, secretory granules, Golgi’s bodies and mitochondrias in cytoplasms. × 7200.

Figure 10 The normal epithelial secretory cells are relatively small, nuclei are round, a great deal of spherical incretory granules in cytoplasms. × 14000.

Figure 11 Normal gastric mucosa epithelial nuclei are round. or oval, nucleocyotoplasmic ratio < 1; the nucleoli have no capsules. × 14000.

Figure 12 Normal cristas are hollow canals leading to outer ventricles. cristas are vertical to mitochondrial long axles. × 14000.

Figure 13 Intercellular space expands, decrease of cellular conjuctions; mitochondria decrease, swollen or cristae break, have vacuolar degeneration. × 29000.

Figure 14 Nucleocytoplasmic ratio > 1, lobulated nuclei. × 7200.

Figure 15 Heterochromatins lie densely around nuclei, the electron density is low in the centers of nuclei. the nuclei are loop in shape (referred to as chromatic homogenization). × 14000.

Figure 16 Longitudinal cristas, sparse cristae, deranged cristae. × 14000.