Comparative study on the pathogenesis of Crohn’s disease and ulcerative colitis

doi:10.3748/wjg.v31.i19.106406

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May 21, 2025 (publication date) through May 21, 2025

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. May 21, 2025; 31(19): 106406
Published online May 21, 2025. doi: 10.3748/wjg.v31.i19.106406

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Figure 1 Study on the pathogenesis between Crohn’s disease and ulcerative colitis. IBD: Inflammatory bowel disease.

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Figure 2 Key cytokines and their functions of immune system for the pathogenesis of inflammatory bowel disease.

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Figure 3 Gene function changes for the pathogenesis of inflammatory bowel disease. ATG16L1: Autophagy-related 16-like 1; CARD9: Caspase-recruitment domain 9; CCR6: CC-motif chemokine receptor 6; ELF1: Elongation factor 1; FCHSD2: FCH and double SH3 domains protein 2; FGFR1OP: Fibroblast growth factor receptor 1 oncogene partner; IL: Interleukin; IRGM: Immunity-related GTPase M; JAK2: Janus kinase 2; NKX2-3: NK2 transcription factor related, locus 3; NOD2: Nucleotide-binding oligomerization domain 2; NUDT15: Nudix hydrolase 15; PRDM1: PR domain zinc finger protein 1; SLC25A15: Solute carrier family 25 member 15; STAT3: Signal transducer and activator of transcription 3; TNFSF15: Tumor necrosis family superfamily member 15; WBP4: WW domain-binding protein 4.

Citation: Yang QH, Zhang CN. Comparative study on the pathogenesis of Crohn’s disease and ulcerative colitis. World J Gastroenterol 2025; 31(19): 106406
URL: https://www.wjgnet.com/1007-9327/full/v31/i19/106406.htm
DOI: https://dx.doi.org/10.3748/wjg.v31.i19.106406