COVID-19, liver dysfunction and pathophysiology: A conceptual discussion

doi:10.3748/wjg.v28.i6.683

Advanced Search

BPG is committed to discovery and dissemination of knowledge

Home / Archive / Volume 28, Issue 6

This Article

Academic Content and Language Evaluation of This Article

Academic Rules and Norms of This Article

Citation of this article

Corresponding Author of This Article

Research Domain of This Article

Article-Type of This Article

Open-Access Policy of This Article

Times Cited Counts in Google of This Article

Number of Hits and Downloads for This Article

Total Article Views (4000)

All Articles published online

The chart showing PDF series, WORD series, HTML series, Figures (1-1) series.

Item

Count

PDF

271

WORD

184

HTML

2089

Figures (1-1)

298

Sum=2842

Publishing Process of This Article

The chart showing Browse series, Download series.

Item

Count

Browse

378

Download

780

Sum=1158

Feb 14, 2022 (publication date) through Jul 25, 2024

Times Cited of This Article

Times Cited (3)

Journal Information of This Article

Publication Name

World Journal of Gastroenterology

ISSN

1007-9327

Publisher of This Article

Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Letter to the Editor

World J Gastroenterol. Feb 14, 2022; 28(6): 683-688
Published online Feb 14, 2022. doi: 10.3748/wjg.v28.i6.683

Open in New Tab Full Size Figure Download Figure

Figure 1 Pathophysiological hypotheses explaining liver injury by severe acute respiratory syndrome coronavirus-2. ACE2: Angiotensin II; ADAM17: AT1R-metalloprotease 17; CD147: Cluster of Differentiation 147; L-SIGN: Liver/Lymph node-specific intercellular adhesion molecule-3-grabbing non-integrin.

Citation: Gonçalves Júnior J. COVID-19, liver dysfunction and pathophysiology: A conceptual discussion. World J Gastroenterol 2022; 28(6): 683-688
URL: https://www.wjgnet.com/1007-9327/full/v28/i6/683.htm
DOI: https://dx.doi.org/10.3748/wjg.v28.i6.683