Observational Study
Copyright ©The Author(s) 2020.
World J Gastroenterol. Nov 28, 2020; 26(44): 7046-7060
Published online Nov 28, 2020. doi: 10.3748/wjg.v26.i44.7046
Figure 1
Figure 1 Flow chart displaying the selection of participants in the study cohort. PCOS: Polycystic ovary syndrome; TE: Transient elastography.
Figure 2
Figure 2 Distribution of metabolically normal and abnormal patients by nonalcoholic fatty liver disease category. NAFLD: Nonalcoholic fatty liver disease.
Figure 3
Figure 3 Prevalence of nonalcoholic fatty liver disease and significant liver fibrosis. A: Prevalence of nonalcoholic fatty liver disease (NAFLD), severe NAFLD and significant liver fibrosis according to body mass index category; and B: Prevalence of NAFLD according to patients’ characteristics. NAFLD: Nonalcoholic fatty liver disease; ALT: Alanine aminotransferase; HOMA-IR: Homeostasis model for assessment of insulin resistance.
Figure 4
Figure 4 Scatterplot depicting the correlation between liver stiffness measurement. A: Aspartate aminotransferase-to-Platelets Ratio Index; B: Fibrosis-4; and C: Nonalcoholic fatty liver disease fibrosis score. NAFLD: Nonalcoholic fatty liver disease; APRI: aspartate aminotransferase-to-Platelets Ratio Index; FIB-4: Fibrosis-4.
Figure 5
Figure 5 Area under the curve of body mass index, free androgen index and alanine aminotransferase for prediction of nonalcoholic fatty liver disease. BMI: Body mass index; ALT: Alanine aminotransferase; AUC: Area under the curve; FAI: Free androgen index.