Retrospective Study
Copyright ©The Author(s) 2019.
World J Gastroenterol. Mar 21, 2019; 25(11): 1398-1408
Published online Mar 21, 2019. doi: 10.3748/wjg.v25.i11.1398
Figure 1
Open in New Tab Full Size Figure Download Figure
Figure 1 Patient disposition. CHB: Chronic hepatitis B; HCC: Hepatocellular carcinoma; HCV: Hepatitis C virus; HBeAg: Hepatitis B e antigen; HIV: Human immunodeficiency virus.
Figure 2
Open in New Tab Full Size Figure Download Figure
Figure 2 Serum Mac-2 binding protein glycosylation isomer levels at baseline, 5 years and 10 years after hepatitis B e antigen seroconversion according to presence of cirrhosis. Boxplot: Horizontal lines, colored bars and error bars represent median, interquartile range and 95% confidence interval, respectively. M2BPGi: Mac-2 binding protein glycosylation isomer.
Figure 3
Open in New Tab Full Size Figure Download Figure
Figure 3 Serum Mac-2 binding protein glycosylation isomer levels at baseline, 5 years and 10 years after hepatitis B e antigen seroconversion in patients with or without subsequent development of hepatocellular carcinoma. Boxplot: Horizontal lines, colored bars and error bars represent median, interquartile range and 95% confidence interval, respectively. HCC: Hepatocellular carcinoma; M2BPGi: Mac-2 binding protein glycosylation isomer.
Figure 4
Open in New Tab Full Size Figure Download Figure
Figure 4 Cumulative incidence of hepatocellular carcinoma according to baseline serum Mac-2 binding protein glycosylation isomer level. COI: Cut-off index; HCC: Hepatocellular carcinoma; M2BPGi: Mac-2 binding protein glycosylation isomer.