Potential mechanisms of hepatitis B virus induced liver injury

doi:10.3748/wjg.v20.i35.12462

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606

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Sep 21, 2014 (publication date) through Jul 22, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Sep 21, 2014; 20(35): 12462-12472
Published online Sep 21, 2014. doi: 10.3748/wjg.v20.i35.12462

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Figure 1 Mitochondrial, nuclear and cytoplasmic pathways activated by HBx. EMT: Epithelial-mesenchymal transition; TERT: Human telomerase reverse transcriptase; TGFβ: Transforming Growth Factor-β; UPR: Unfolded Protein Response; HDAC: Histone Deacetylase; FN: Fibronectin; ROS: Reactive Oxygen Species; NF-Κb: Nuclear Factor-κB; A2Mα: 2-Macroglobulin; hVDAC: Voltage-Dependent Anion Channel; CDH1: E-cadherin; ER: Endoplasmic reticulum; NF-AT: Nuclear factor of activated T-cells; ATF: Activated transcription factor peroxisome; PPAR-γ: Proliferator-activated receptor gamma; AP: Transcription activitor.

Citation: Suhail M, Abdel-Hafiz H, Ali A, Fatima K, Damanhouri GA, Azhar E, Chaudhary AG, Qadri I. Potential mechanisms of hepatitis B virus induced liver injury. World J Gastroenterol 2014; 20(35): 12462-12472
URL: https://www.wjgnet.com/1007-9327/full/v20/i35/12462.htm
DOI: https://dx.doi.org/10.3748/wjg.v20.i35.12462