Significance of regenerating islet-derived type IV gene expression in gastroenterological cancers

doi:10.3748/wjg.v18.i27.3502

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Jul 21, 2012 (publication date) through May 1, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Jul 21, 2012; 18(27): 3502-3510
Published online Jul 21, 2012. doi: 10.3748/wjg.v18.i27.3502

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Figure 1 Schematic representation of the regenerating islet-derived type IV signaling pathway. Regenerating islet-derived type IV (RegIV) contributes to cell proliferation, inhibition of apoptosis, and carcinogenesis via protein kinase B (Akt) signaling pathway by transactivating the epidermal growth factor receptor (EGFR). The primary signaling pathway is now being investigated. MBS: Mannan-binding sites; PI3K: Phosphoinositide 3-kinase; PIP2: Phosphatidylinositol 4,5-bisphosphate; PIP3: Phosphatidylinositol 4,5-triphosphate; PDK1: Phosphoinositide-dependent kinase-1; Bcl-2: B-cell lymphoma 2.

Citation: Numata M, Oshima T. Significance of regenerating islet-derived type IV gene expression in gastroenterological cancers. World J Gastroenterol 2012; 18(27): 3502-3510
URL: https://www.wjgnet.com/1007-9327/full/v18/i27/3502.htm
DOI: https://dx.doi.org/10.3748/wjg.v18.i27.3502