Original Article
Copyright ©2011 Baishideng Publishing Group Co.
World J Gastroenterol. Jan 21, 2011; 17(3): 322-328
Published online Jan 21, 2011. doi: 10.3748/wjg.v17.i3.322
Figure 1
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Figure 1 Representative photomicrographs of rats in cirrhotic and sham groups (magnification 200 ×, HE staining). A: Normal liver structure; B: Liver cirrhosis; C: Normal kidney structure; D: Renal structure in cirrhotic group.
Figure 2
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Figure 2 Expression of heme oxygenase-1 mRNA in kidney. A: Representative reverse-transcription polymerase chain reaction data showing the heme oxygenase-1 (HO-1) mRNA expression levels in kidneys from zinc protoporphyrin IX (ZnPP) treatment group (lane 1), cirrhotic group (lane 2), cobalt protoporphyrin (CoPP) treatment group (lane 3), and sham group (lane 4); B: Quantitative data showing the ratio of band density of the corresponding HO-1 mRNA to that of β-actin mRNA.
Figure 3
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Figure 3 Expression of heme oxygenase-1 protein in kidney and liver. Immunohistochemical staining of renal heme oxygenase-1 (HO-1) protein in rats of sham group (A), cirrhotic group (B), zinc protoporphyrin IX (ZnPP) treatment group (C), cobalt protoporphyrin (CoPP) treatment group (D), and immunohistochemical staining of hepatic HO-1 protein in rats of sham group (E) and cirrhotic group (F) in the upper part (magnification 200 ×), and quantitative scoring (G) of immunohistochemical staining of renal HO-1 protein expression in each group in the lower part.