Original Articles
Copyright ©2009 The WJG Press and Baishideng.
World J Gastroenterol. Sep 28, 2009; 15(36): 4529-4537
Published online Sep 28, 2009. doi: 10.3748/wjg.15.4529
Figure 1
Open in New Tab Full Size Figure Download Figure
Figure 1 Histological fingdings of liver tissue of the rats in different experimental groups. A: Normal healthy rats; B: LC rats; C: NTau-treated groups. (magnification, 10 × 20).
Figure 2
Open in New Tab Full Size Figure Download Figure
Figure 2 Bar chart presentation of the amount of COL I, COL III and TGF-β1 in the liver tissue of rats in different experimental groups.
Figure 3
Open in New Tab Full Size Figure Download Figure
Figure 3 Immunohistochemical analysis of the expression of COL I in liver tissue of rats in different experimental groups. A: Normal healthy rats; B: LC Rats. Note the expression of a large amount of COL I; C: NTau-treated rats. Note the reduction of the expression of COL I. (magnification, 10 × 20).
Figure 4
Open in New Tab Full Size Figure Download Figure
Figure 4 Immunohistochemical analysis of the expression of COL III in liver tissue of rats in different experimental groups. A: Normal healthy rats; B: LC rats. Note the abundance in the expression of COL III; C: NTau-treated rats. The expression of COL III was decreased. (magnification, 10 × 20).
Figure 5
Open in New Tab Full Size Figure Download Figure
Figure 5 Immunohistochemical analysis of the expression of TGF-β1 in liver tissue of rats in different experimental groups. A: Normal healthy rats; B: LC rats. Note the increased expression of TGF-β1; C: NTau-treated rats. Note that the expression of TGF-β1 was reduced. (magnification, 10 × 20).
Figure 6
Open in New Tab Full Size Figure Download Figure
Figure 6 Comparison of the portal ring compliance among rats in different experimental groups.