Rapid Communication
Copyright ©2008 The WJG Press and Baishideng.
World J Gastroenterol. Sep 21, 2008; 14(35): 5461-5466
Published online Sep 21, 2008. doi: 10.3748/wjg.14.5461
Figure 1
Figure 1 A: Effect of 10 nmol/L LY83583 on cGMP production of in tissue (n = 8), bP < 0. 01 vs control group, dP < 0.01 vs DNP group; B: Effect of 10 nmol/L LY83583 on cGMP production of in efflux (n = 8), fP < 0.01 vs control group, hP < 0.01 vs DNP group; C: Effect of a membrane-permeable PKG-specific inhibitor (KT5823) on DNP-induced inhibition of spontaneous contraction in gastric antral circular smooth muscle (n = 8), jP < 0.01 vs DNP group.
Figure 2
Figure 2 Effect of LY83583 on DNP-induced increase of IK(Ca), bP < 0. 01 vs control group.
Figure 3
Figure 3 Effect of a membrane-permeable PKG-specific inhibitor, KT-5823, on DNP-induced increase of IK(Ca) (n = 8), bP < 0. 01 vs control group.
Figure 4
Figure 4 A: Effect of a membrane-permeable PKG-specific inhibitor, KT-5823, on STOCs (n = 10); B: Effect of KT-5720 (500 nmol/L), a PKA-specific inhibitor, on STOCs (n = 10).