Possible mechanism for the regulation of glucose on proliferation, inhibition and apoptosis of colon cancer cells induced by sodium butyrate

Advanced Search

BPG is committed to discovery and dissemination of knowledge

Home / Archive / Volume 13, Issue 29

This Article

Citation of this article

Corresponding Author of This Article

Article-Type of This Article

Open-Access Policy of This Article

Times Cited Counts in Google of This Article

Number of Hits and Downloads for This Article

All Articles published online

Item

Count

PDF

208

HTML

1318

Figures (1-3)

235

Sum=1761

Aug 7, 2007 (publication date) through Dec 22, 2024

Times Cited of This Article

Journal Information of This Article

Publication Name

World Journal of Gastroenterology

ISSN

1007-9327

Publisher of This Article

Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Rapid Communication

World J Gastroenterol. Aug 7, 2007; 13(29): 4015-4018
Published online Aug 7, 2007. doi: 10.3748/wjg.v13.i29.4015

Figure 1 The survival rate of HT-29 cell. ^aP < 0.05 vs without sodium butyrate.

Figure 2 The expression of GluT1 and of MCT1 of HT-29 cell decreased with the increase of glucose concentration.

Figure 3 HT-29 cancer cell apoptosis (TUNEL, × 400). A: 2000 mg/L glucose; B: Without glucose; C: 5 mmol/L sodium butyrate.

Citation: He L, Li X, Luo HS, Rong H, Cai J. Possible mechanism for the regulation of glucose on proliferation, inhibition and apoptosis of colon cancer cells induced by sodium butyrate. World J Gastroenterol 2007; 13(29): 4015-4018
URL: https://www.wjgnet.com/1007-9327/full/v13/i29/4015.htm
DOI: https://dx.doi.org/10.3748/wjg.v13.i29.4015