Possible mechanism for the regulation of glucose on proliferation, inhibition and apoptosis of colon cancer cells induced by sodium butyrate

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Aug 7, 2007 (publication date) through Nov 20, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Aug 7, 2007; 13(29): 4015-4018
Published online Aug 7, 2007. doi: 10.3748/wjg.v13.i29.4015

Figure 1 The survival rate of HT-29 cell. ^aP < 0.05 vs without sodium butyrate.

Figure 2 The expression of GluT1 and of MCT1 of HT-29 cell decreased with the increase of glucose concentration.

Figure 3 HT-29 cancer cell apoptosis (TUNEL, × 400). A: 2000 mg/L glucose; B: Without glucose; C: 5 mmol/L sodium butyrate.

Citation: He L, Li X, Luo HS, Rong H, Cai J. Possible mechanism for the regulation of glucose on proliferation, inhibition and apoptosis of colon cancer cells induced by sodium butyrate. World J Gastroenterol 2007; 13(29): 4015-4018
URL: https://www.wjgnet.com/1007-9327/full/v13/i29/4015.htm
DOI: https://dx.doi.org/10.3748/wjg.v13.i29.4015