Epigenetic effects of ethanol on liver and gastrointestinal injury

doi:10.3748/wjg.v12.i33.5265

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Sep 7, 2006 (publication date) through Jan 13, 2025

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Sep 7, 2006; 12(33): 5265-5271
Published online Sep 7, 2006. doi: 10.3748/wjg.v12.i33.5265

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Figure 1 A diagram depicting relationship among ethanol metabolic stress, epigenetics and tissue injury. Acet: acetaldehyde; ALD: alcoholic liver disease; DNMT: DNA methyl transferases; ER: endoplasmic reticulum; FAEE: fatty acyl ethyl esters; GSH: glutathione; HAT: histone acetyl transferases; HCV: hepatitis C virus; HDAC: histone deacetylase; HMT: histone methyl transferases; PEth: phosphatidylethanol; ROS: reactive oxygen species; SAM: S-adenosylmethionine.

Citation: Shukla SD, Aroor AR. Epigenetic effects of ethanol on liver and gastrointestinal injury. World J Gastroenterol 2006; 12(33): 5265-5271
URL: https://www.wjgnet.com/1007-9327/full/v12/i33/5265.htm
DOI: https://dx.doi.org/10.3748/wjg.v12.i33.5265