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©2005 Baishideng Publishing Group Inc.
World J Gastroenterol. Jun 28, 2005; 11(24): 3675-3679
Published online Jun 28, 2005. doi: 10.3748/wjg.v11.i24.3675
Published online Jun 28, 2005. doi: 10.3748/wjg.v11.i24.3675
Figure 1 Results of insulin resistance sensitivity assay in hyperinsulinemic-euglycemic clamp test (A) and changes of glucose induced by insulin injection (B).
NC: control diet group; FD: fat emulsion diet group; GIR: glucose injection rate. aP<0.05, bP<0.01 vs NC.
Figure 2 Morphological observation of fat and pancreas islets stained HE in rats with insulin resistance (60×).
A: Fat cells in control group; B: fat cells in high fat emulsion group; C: pancreas islets in control group; D: pancreas islets in high fat emulsion group.
Figure 3 mRNA expression assay of GLUT2 and α-glucosidase in small intestinal epithelium and GLUT4 in skeletal muscle in rats with insulin resistance (30×).
A: GLUT2 in control group; B: GLUT2 in high fat emulsion group; C: α-glucosidase in control group; D: α-glucosidase in high fat emulsion group; E: GLUT4 in control group; F: GLUT4 in high fat emulsion group.
- Citation: Ai J, Wang N, Yang M, Du ZM, Zhang YC, Yang BF. Development of Wistar rat model of insulin resistance. World J Gastroenterol 2005; 11(24): 3675-3679
- URL: https://www.wjgnet.com/1007-9327/full/v11/i24/3675.htm
- DOI: https://dx.doi.org/10.3748/wjg.v11.i24.3675