Analysis of the mechanisms of rabbit’s brainstem hemorrhage complicated with irritable changes in the alvine mucous membrane

Figure 1 Changes in the pressure of the lateral ventricle during acute brainstem hemorrhage in rabbit.

Figure 2 Changes in caliber of jejunal mesentery small vessels under the load of rabbit’s brainstem hemorrhage.

Figure 3 Changes of microcirculation of jejunal mucosa. A: Photograph of microcirculation of jejunal mucous membrane, 6 s before the load of rabbit’s brainstem hemorrhage (×185); B: Photograph of jejunal mucous membrane, 10 min 26 s after the load of rabbit’s brainstem hemorrhage; it showed the local congestion of villus mucosa (×185); C: Photograph of jejunal mucous membrane, 30 min 32 s after the load of rabbit’s brainstem hemorrhage; it showed that the local congestion of villus mucosa had relieved (×185); D: Photograph of jejunal mucous membrane, 40 min 46 s after the load of rabbit’s brainstem hemorrhage; it showed that the local congestion of villus mucosa had ameliorated obviously (×185).

Figure 4 Paired t-test for red color coordinate mean values of jejunal mucous membrane before and after rabbit’s brainstem hemorrhage.

Figure 5 Comparison of electric discharge of vagus nerve. A: The comparison of the electric discharge of vagus nerve through analyzing the absolute value of an area of wave form before and after the compression (from 4 to 40 s under the intracranial hypertension), and the neural discharge of vagus nerve had increased after intracranial hypertension, n = 20, P<0.05; B: The comparison of the electric discharge of vagus nerve before and during the first 4 s after the rabbit’s lateral ventricle compression. We analyzed the electric discharge frequency of the dischargeable wave in each time segment by setting a single liminal value line, and the neural discharge of vagus nerve had increased after intracranial hypertension; C: The comparison of the electric discharge of vagus nerve before the compression and during continuous compression (from 4 to 40 s). We analyzed the electric discharge frequency of the dischargeable wave in each time segment by setting a single liminal value line, and the neural discharge of vagus nerve had increased after intracranial hypertension.

Figure 6 HE stain of duodenal structure of rabbits. A: HE stain of normal rabbit’s duodenal villus. The minute blood vessel had not dilated and red blood cells had not leaked out from capillary lumens; B: HE stain of normal rabbit’s mucous glands of duodenum. The minute blood vessel had not dilated; C: HE stain of the rabbit’s duodenal villus, which were 48 h after brainstem hemorrhage. The capillaries had obviously dilated and red blood cells had leaked out from capillary lumens; D: HE stain of the rabbit’s mucous glands of duodenum, which were 48 h after brainstem hemorrhage. The capillaries had obviously dilated and red blood cells had leaked out from capillary lumens.