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For: Li C, Kuemmerle JF. Genetic and epigenetic regulation of intestinal fibrosis. United European Gastroenterol J 2016;4:496-505. [PMID: 27536359 DOI: 10.1177/2050640616659023] [Cited by in Crossref: 4] [Cited by in F6Publishing: 4] [Article Influence: 0.7] [Reference Citation Analysis]
Number Citing Articles
1 Li C, Kuemmerle JF. The fate of myofibroblasts during the development of fibrosis in Crohn's disease. J Dig Dis 2020;21:326-31. [PMID: 32092217 DOI: 10.1111/1751-2980.12852] [Cited by in Crossref: 6] [Cited by in F6Publishing: 5] [Article Influence: 6.0] [Reference Citation Analysis]
2 Latella G. Redox Imbalance in Intestinal Fibrosis: Beware of the TGFβ-1, ROS, and Nrf2 Connection. Dig Dis Sci 2018;63:312-20. [DOI: 10.1007/s10620-017-4887-1] [Cited by in Crossref: 21] [Cited by in F6Publishing: 22] [Article Influence: 4.2] [Reference Citation Analysis]
3 Li C, Grider JR, Murthy KS, Bohl J, Rivet E, Wieghard N, Kuemmerle JF. Endoplasmic Reticulum Stress in Subepithelial Myofibroblasts Increases the TGF-β1 Activity That Regulates Fibrosis in Crohn's Disease. Inflamm Bowel Dis 2020;26:809-19. [PMID: 32031621 DOI: 10.1093/ibd/izaa015] [Cited by in Crossref: 11] [Cited by in F6Publishing: 9] [Article Influence: 11.0] [Reference Citation Analysis]
4 Li C. Unfolded Protein Response and Crohn’s Diseases: A Molecular Mechanism of Wound Healing in the Gut. GastrointestDisord 2021;3:31-43. [DOI: 10.3390/gidisord3010004] [Cited by in Crossref: 2] [Article Influence: 2.0] [Reference Citation Analysis]
5 Rieder F. Toward an antifibrotic therapy for inflammatory bowel disease. United European Gastroenterol J 2016;4:493-5. [PMID: 27536358 DOI: 10.1177/2050640616660000] [Cited by in Crossref: 8] [Cited by in F6Publishing: 9] [Article Influence: 1.3] [Reference Citation Analysis]