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Cited by in F6Publishing
For: Ogata S, Hashizume K, Hayase Y, Kanno Y, Hori K, Balan S, Yoshikawa T, Takahashi H, Taya S, Hoshino M. Potential involvement of DSCAML1 mutations in neurodevelopmental disorders. Genes Cells 2021;26:136-51. [PMID: 33501714 DOI: 10.1111/gtc.12831] [Cited by in Crossref: 3] [Cited by in F6Publishing: 3] [Article Influence: 1.5] [Reference Citation Analysis]
Number Citing Articles
1 Ma M, Brunal AA, Clark KC, Studtmann C, Stebbins K, Higashijima SI, Pan YA. Deficiency in the cell-adhesion molecule dscaml1 impairs hypothalamic CRH neuron development and perturbs normal neuroendocrine stress axis function. Front Cell Dev Biol 2023;11:1113675. [PMID: 36875755 DOI: 10.3389/fcell.2023.1113675] [Reference Citation Analysis]
2 Ma M, Brunal AA, Clark KC, Studtmann C, Stebbins K, Higashijima S, Pan YA. Deficiency in the cell-adhesion molecule dscaml1 impairs hypothalamic CRH neuron development and neuroendocrine stress axis function.. [DOI: 10.1101/2022.09.22.509087] [Reference Citation Analysis]
3 Wang Q, Bi Y, Wang Z, Zhu H, Liu M, Wu X, Pan C. Goat SNX29: mRNA expression, InDel and CNV detection, and their associations with litter size. Front Vet Sci 2022;9:981315. [DOI: 10.3389/fvets.2022.981315] [Reference Citation Analysis]
4 Szpirer C. Rat Models of Human Diseases and Related Phenotypes: A Novel Inventory of Causative Genes. Mamm Genome 2021. [PMID: 34184128 DOI: 10.1007/s00335-021-09876-2] [Cited by in Crossref: 1] [Article Influence: 0.5] [Reference Citation Analysis]
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