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For: Kravtsova-Ivantsiv Y, Goldhirsh G, Ivantsiv A, Ben Itzhak O, Kwon YT, Pikarsky E, Ciechanover A. Excess of the NF-ĸB p50 subunit generated by the ubiquitin ligase KPC1 suppresses tumors via PD-L1- and chemokines-mediated mechanisms. Proc Natl Acad Sci U S A 2020;117:29823-31. [PMID: 33168738 DOI: 10.1073/pnas.2019604117] [Cited by in Crossref: 8] [Cited by in F6Publishing: 9] [Article Influence: 4.0] [Reference Citation Analysis]
Number Citing Articles
1 Cavazos TB, Kachuri L, Graff RE, Nierenberg JL, Thai KK, Alexeeff S, Van Den Eeden S, Corley DA, Kushi LH, Hoffmann TJ, Ziv E, Habel LA, Jorgenson E, Sakoda LC, Witte JS; Regeneron Genetics Center. Assessment of genetic susceptibility to multiple primary cancers through whole-exome sequencing in two large multi-ancestry studies. BMC Med 2022;20:332. [PMID: 36199081 DOI: 10.1186/s12916-022-02535-6] [Reference Citation Analysis]
2 Hosseiniyan Khatibi SM, Ardalan M, Teshnehlab M, Vahed SZ, Pirmoradi S. Panels of mRNAs and miRNAs for decoding molecular mechanisms of Renal Cell Carcinoma (RCC) subtypes utilizing Artificial Intelligence approaches. Sci Rep 2022;12:16393. [PMID: 36180558 DOI: 10.1038/s41598-022-20783-7] [Reference Citation Analysis]
3 Cavazos TB, Kachuri L, Graff RE, Nierenberg JL, Thai KK, Alexeeff S, Van Den Eeden S, Corley DA, Kushi LH, Hoffmann TJ, Ziv E, Habel L, Jorgenson E, Sakoda LC, Witte JS, Regeneron Genetics Center. Assessment of Genetic Susceptibility to Multiple Primary Cancers through Whole-Exome Sequencing in Two Large Multi-Ancestry Studies.. [DOI: 10.1101/2022.02.11.22270688] [Reference Citation Analysis]
4 Goldhirsh G, Kravtsova-Ivantsiv Y, Satish G, Ziv T, Brik A, Ciechanover A. A short binding site in the KPC1 ubiquitin ligase mediates processing of NF-κB1 p105 to p50: A potential for a tumor-suppressive PROTAC. Proc Natl Acad Sci U S A 2021;118:e2117254118. [PMID: 34873064 DOI: 10.1073/pnas.2117254118] [Reference Citation Analysis]
5 Liu FJ, Gu TJ, Wei DY. Emodin alleviates sepsis-mediated lung injury via inhibition and reduction of NF-kB and HMGB1 pathways mediated by SIRT1. Kaohsiung J Med Sci 2021. [PMID: 34806822 DOI: 10.1002/kjm2.12476] [Cited by in Crossref: 1] [Cited by in F6Publishing: 1] [Article Influence: 1.0] [Reference Citation Analysis]
6 Delvecchio VS, Fierro C, Giovannini S, Melino G, Bernassola F. Emerging roles of the HECT-type E3 ubiquitin ligases in hematological malignancies. Discov Onc 2021;12. [DOI: 10.1007/s12672-021-00435-4] [Cited by in Crossref: 1] [Cited by in F6Publishing: 1] [Article Influence: 1.0] [Reference Citation Analysis]
7 Zhang S, Gao J, Liu S, Yu L, Zhang W, Liang Y, Wang H. Transcription Coactivator BCL3 Acts as a Potential Regulator of Lipid Metabolism Through the Effects on Inflammation. J Inflamm Res 2021;14:4915-26. [PMID: 34588797 DOI: 10.2147/JIR.S327858] [Cited by in Crossref: 2] [Cited by in F6Publishing: 2] [Article Influence: 2.0] [Reference Citation Analysis]
8 Bailly C. Anticancer Targets and Signaling Pathways Activated by Britannin and Related Pseudoguaianolide Sesquiterpene Lactones. Biomedicines 2021;9:1325. [PMID: 34680439 DOI: 10.3390/biomedicines9101325] [Cited by in Crossref: 3] [Cited by in F6Publishing: 3] [Article Influence: 3.0] [Reference Citation Analysis]
9 Rosell R, Gonzalez-Cao M. Cemiplimab monotherapy in advanced non-squamous and squamous non-small cell lung cancer. Lancet 2021;397:557-9. [PMID: 33581807 DOI: 10.1016/S0140-6736(21)00196-3] [Cited by in Crossref: 3] [Cited by in F6Publishing: 3] [Article Influence: 3.0] [Reference Citation Analysis]