BPG is committed to discovery and dissemination of knowledge
Cited by in F6Publishing
For: Cubria MB, Suarez S, Masoudi A, Oftadeh R, Kamalapathy P, DuBose A, Erdos MR, Cabral WA, Karim L, Collins FS, Snyder BD, Nazarian A. Evaluation of musculoskeletal phenotype of the G608G progeria mouse model with lonafarnib, pravastatin, and zoledronic acid as treatment groups. Proc Natl Acad Sci U S A 2020;117:12029-40. [PMID: 32404427 DOI: 10.1073/pnas.1906713117] [Cited by in Crossref: 8] [Cited by in F6Publishing: 10] [Article Influence: 4.0] [Reference Citation Analysis]
Number Citing Articles
1 Trani JP, Chevalier R, Caron L, El Yazidi C, Broucqsault N, Toury L, Thomas M, Annab K, Binetruy B, De Sandre-giovannoli A, Levy N, Magdinier F, Robin JD. Mesenchymal stem cells derived from patients with premature aging syndromes display hallmarks of physiological aging. Life Sci Alliance 2022;5:e202201501. [DOI: 10.26508/lsa.202201501] [Reference Citation Analysis]
2 Mosevitsky MI. Progerin and Its Role in Accelerated and Natural Aging. Mol Biol 2022;56:125-46. [DOI: 10.1134/s0026893322020091] [Reference Citation Analysis]
3 Teissier T, Temkin V, Pollak RD, Cox LS. Crosstalk Between Senescent Bone Cells and the Bone Tissue Microenvironment Influences Bone Fragility During Chronological Age and in Diabetes. Front Physiol 2022;13:812157. [DOI: 10.3389/fphys.2022.812157] [Cited by in Crossref: 1] [Cited by in F6Publishing: 1] [Article Influence: 1.0] [Reference Citation Analysis]
4 Wang Y, Shilagardi K, Hsu T, Odinammadu KO, Maruyama T, Wu W, Lin CS, Damoci CB, Spear ED, Shin JY, Hsu W, Michaelis S, Worman HJ. Abolishing the prelamin A ZMPSTE24 cleavage site leads to progeroid phenotypes with near-normal longevity in mice. Proc Natl Acad Sci U S A 2022;119:e2118695119. [PMID: 35197292 DOI: 10.1073/pnas.2118695119] [Reference Citation Analysis]
5 Cabral WA, Tavarez UL, Beeram I, Yeritsyan D, Boku YD, Eckhaus MA, Nazarian A, Erdos MR, Collins FS. Genetic reduction of mTOR extends lifespan in a mouse model of Hutchinson-Gilford Progeria syndrome. Aging Cell 2021;20:e13457. [PMID: 34453483 DOI: 10.1111/acel.13457] [Cited by in Crossref: 4] [Cited by in F6Publishing: 8] [Article Influence: 4.0] [Reference Citation Analysis]
6 Macicior J, Marcos-Ramiro B, Ortega-Gutiérrez S. Small-Molecule Therapeutic Perspectives for the Treatment of Progeria. Int J Mol Sci 2021;22:7190. [PMID: 34281245 DOI: 10.3390/ijms22137190] [Cited by in F6Publishing: 3] [Reference Citation Analysis]
7 Herrmann J, Gummi MR, Xia M, van der Giet M, Tölle M, Schuchardt M. Vascular Calcification in Rodent Models-Keeping Track with an Extented Method Assortment. Biology (Basel) 2021;10:459. [PMID: 34067504 DOI: 10.3390/biology10060459] [Cited by in Crossref: 1] [Cited by in F6Publishing: 2] [Article Influence: 1.0] [Reference Citation Analysis]
8 Benedicto I, Dorado B, Andrés V. Molecular and Cellular Mechanisms Driving Cardiovascular Disease in Hutchinson-Gilford Progeria Syndrome: Lessons Learned from Animal Models. Cells 2021;10:1157. [PMID: 34064612 DOI: 10.3390/cells10051157] [Cited by in F6Publishing: 2] [Reference Citation Analysis]
9 Lu H, Zhang J, Chen YE, Garcia-Barrio MT. Integration of Transformative Platforms for the Discovery of Causative Genes in Cardiovascular Diseases. Cardiovasc Drugs Ther 2021;35:637-54. [PMID: 33856594 DOI: 10.1007/s10557-021-07175-1] [Cited by in Crossref: 1] [Cited by in F6Publishing: 1] [Article Influence: 1.0] [Reference Citation Analysis]
10 Erdos MR, Cabral WA, Tavarez UL, Cao K, Gvozdenovic-Jeremic J, Narisu N, Zerfas PM, Crumley S, Boku Y, Hanson G, Mourich DV, Kole R, Eckhaus MA, Gordon LB, Collins FS. A targeted antisense therapeutic approach for Hutchinson-Gilford progeria syndrome. Nat Med 2021;27:536-45. [PMID: 33707773 DOI: 10.1038/s41591-021-01274-0] [Cited by in Crossref: 8] [Cited by in F6Publishing: 17] [Article Influence: 8.0] [Reference Citation Analysis]