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For: Léna I, Mantegazza M. NaV1.2 haploinsufficiency in Scn2a knock-out mice causes an autistic-like phenotype attenuated with age. Sci Rep 2019;9:12886. [PMID: 31501495 DOI: 10.1038/s41598-019-49392-7] [Cited by in Crossref: 10] [Cited by in F6Publishing: 10] [Article Influence: 3.3] [Reference Citation Analysis]
Number Citing Articles
1 Ma Z, Eaton M, Liu Y, Zhang J, Chen X, Tu X, Shi Y, Que Z, Wettschurack K, Zhang Z, Shi R, Chen Y, Kimbrough A, Lanman NA, Schust L, Huang Z, Yang Y. Deficiency of autism-related Scn2a gene in mice disrupts sleep patterns and circadian rhythms. Neurobiol Dis 2022;:105690. [PMID: 35301122 DOI: 10.1016/j.nbd.2022.105690] [Cited by in Crossref: 1] [Article Influence: 1.0] [Reference Citation Analysis]
2 Li SW, Williams ZM, Báez-Mendoza R. Investigating the Neurobiology of Abnormal Social Behaviors. Front Neural Circuits 2021;15:769314. [PMID: 34916912 DOI: 10.3389/fncir.2021.769314] [Reference Citation Analysis]
3 Wang HG, Bavley CC, Li A, Jones RM, Hackett J, Bayleyen Y, Lee FS, Rajadhyaksha AM, Pitt GS. Scn2a severe hypomorphic mutation decreases excitatory synaptic input and causes autism-associated behaviors. JCI Insight 2021;6:150698. [PMID: 34156984 DOI: 10.1172/jci.insight.150698] [Reference Citation Analysis]
4 Valverde A, Dunys J, Lorivel T, Debayle D, Gay AS, Caillava C, Chami M, Checler F. Dipeptidyl peptidase 4 contributes to Alzheimer's disease-like defects in a mouse model and is increased in sporadic Alzheimer's disease brains. J Biol Chem 2021;297:100963. [PMID: 34265307 DOI: 10.1016/j.jbc.2021.100963] [Reference Citation Analysis]
5 Valverde A, Dunys J, Lorivel T, Debayle D, Gay AS, Lacas-Gervais S, Roques BP, Chami M, Checler F. Aminopeptidase A contributes to biochemical, anatomical and cognitive defects in Alzheimer's disease (AD) mouse model and is increased at early stage in sporadic AD brain. Acta Neuropathol 2021;141:823-39. [PMID: 33881611 DOI: 10.1007/s00401-021-02308-0] [Cited by in Crossref: 8] [Cited by in F6Publishing: 9] [Article Influence: 8.0] [Reference Citation Analysis]
6 Mantegazza M, Cestèle S, Catterall WA. Sodium channelopathies of skeletal muscle and brain. Physiol Rev 2021;101:1633-89. [PMID: 33769100 DOI: 10.1152/physrev.00025.2020] [Cited by in Crossref: 2] [Cited by in F6Publishing: 11] [Article Influence: 2.0] [Reference Citation Analysis]
7 Indumathy J, Pruitt A, Gautier NM, Crane K, Glasscock E. Kv1.1 deficiency alters repetitive and social behaviors in mice and rescues autistic-like behaviors due to Scn2a haploinsufficiency. Brain Behav 2021;11:e02041. [PMID: 33484493 DOI: 10.1002/brb3.2041] [Cited by in Crossref: 3] [Cited by in F6Publishing: 2] [Article Influence: 3.0] [Reference Citation Analysis]
8 Eaton M, Zhang J, Ma Z, Park AC, Lietzke E, Romero CM, Liu Y, Coleman ER, Chen X, Xiao T, Que Z, Lai S, Wu J, Lee JH, Palant S, Nguyen HP, Huang Z, Skarnes WC, Koss WA, Yang Y. Generation and basic characterization of a gene-trap knockout mouse model of Scn2a with a substantial reduction of voltage-gated sodium channel Nav 1.2 expression. Genes Brain Behav 2021;20:e12725. [PMID: 33369088 DOI: 10.1111/gbb.12725] [Cited by in Crossref: 2] [Cited by in F6Publishing: 3] [Article Influence: 2.0] [Reference Citation Analysis]
9 Turner TJ, Zourray C, Schorge S, Lignani G. Recent advances in gene therapy for neurodevelopmental disorders with epilepsy. J Neurochem 2021;157:229-62. [PMID: 32880951 DOI: 10.1111/jnc.15168] [Cited by in Crossref: 5] [Cited by in F6Publishing: 10] [Article Influence: 2.5] [Reference Citation Analysis]
10 Kruth KA, Grisolano TM, Ahern CA, Williams AJ. SCN2A channelopathies in the autism spectrum of neuropsychiatric disorders: a role for pluripotent stem cells? Mol Autism 2020;11:23. [PMID: 32264956 DOI: 10.1186/s13229-020-00330-9] [Cited by in Crossref: 6] [Cited by in F6Publishing: 5] [Article Influence: 3.0] [Reference Citation Analysis]
11 [DOI: 10.1101/2020.09.09.289900] [Cited by in Crossref: 3] [Cited by in F6Publishing: 2] [Reference Citation Analysis]