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Cited by in F6Publishing
For: Tripathi A, Spedding M, Schenker E, Didriksen M, Cressant A, Jay TM. Cognition- and circuit-based dysfunction in a mouse model of 22q11.2 microdeletion syndrome: effects of stress. Transl Psychiatry 2020;10:41. [PMID: 32066701 DOI: 10.1038/s41398-020-0687-z] [Cited by in Crossref: 10] [Cited by in F6Publishing: 8] [Article Influence: 5.0] [Reference Citation Analysis]
Number Citing Articles
1 Al-Absi AR, Thambiappa SK, Khan AR, Glerup S, Sanchez C, Landau AM, Nyengaard JR. Df(h22q11)/+ mouse model exhibits reduced binding levels of GABAA receptors and structural and functional dysregulation in the inhibitory and excitatory networks of hippocampus. Mol Cell Neurosci 2022;122:103769. [PMID: 35988854 DOI: 10.1016/j.mcn.2022.103769] [Reference Citation Analysis]
2 Spedding M, Sebban C, Jay TM, Rocher C, Tesolin-decros B, Chazot P, Schenker E, Szénási G, Lévay GI, Megyeri K, Barkóczy J, Hársing LG, Thomson I, Cunningham MO, Whittington MA, Etherington L, Lambert JJ, Antoni FA, Gacsályi I. Phenotypical Screening on Neuronal Plasticity in Hippocampal-Prefrontal Cortex Connectivity Reveals an Antipsychotic with a Novel Profile. Cells 2022;11:1181. [DOI: 10.3390/cells11071181] [Reference Citation Analysis]
3 Hiramoto T, Sumiyoshi A, Yamauchi T, Tanigaki K, Shi Q, Kang G, Ryoke R, Nonaka H, Enomoto S, Izumi T, Bhat MA, Kawashima R, Hiroi N. Tbx1, a gene encoded in 22q11.2 copy number variant, is a link between alterations in fimbria myelination and cognitive speed in mice. Mol Psychiatry 2021. [PMID: 34737458 DOI: 10.1038/s41380-021-01318-4] [Cited by in Crossref: 2] [Cited by in F6Publishing: 2] [Article Influence: 2.0] [Reference Citation Analysis]
4 Kolk SM, Rakic P. Development of prefrontal cortex. Neuropsychopharmacology 2022;47:41-57. [PMID: 34645980 DOI: 10.1038/s41386-021-01137-9] [Cited by in Crossref: 9] [Cited by in F6Publishing: 9] [Article Influence: 9.0] [Reference Citation Analysis]
5 Saito R, Miyoshi C, Koebis M, Kushima I, Nakao K, Mori D, Ozaki N, Funato H, Yanagisawa M, Aiba A. Two novel mouse models mimicking minor deletions in 22q11.2 deletion syndrome revealed the contribution of each deleted region to psychiatric disorders. Mol Brain 2021;14:68. [PMID: 33845872 DOI: 10.1186/s13041-021-00778-7] [Reference Citation Analysis]
6 Spedding M, Chattarji S, Spedding C, Jay TM. Brain circuits at risk in psychiatric diseases and pharmacological pathways. Therapie 2021;76:75-86. [PMID: 33358639 DOI: 10.1016/j.therap.2020.12.005] [Reference Citation Analysis]
7 Arioka Y, Shishido E, Kushima I, Suzuki T, Saito R, Aiba A, Mori D, Ozaki N. Chromosome 22q11.2 deletion causes PERK-dependent vulnerability in dopaminergic neurons. EBioMedicine 2021;63:103138. [PMID: 33341442 DOI: 10.1016/j.ebiom.2020.103138] [Cited by in Crossref: 3] [Cited by in F6Publishing: 4] [Article Influence: 1.5] [Reference Citation Analysis]
8 Al-Absi AR, Qvist P, Okujeni S, Khan AR, Glerup S, Sanchez C, Nyengaard JR. Layers II/III of Prefrontal Cortex in Df(h22q11)/+ Mouse Model of the 22q11.2 Deletion Display Loss of Parvalbumin Interneurons and Modulation of Neuronal Morphology and Excitability. Mol Neurobiol 2020;57:4978-88. [PMID: 32820460 DOI: 10.1007/s12035-020-02067-1] [Reference Citation Analysis]