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Cited by in F6Publishing
For: Zha D, Cheng H, Li W, Wu Y, Li X, Zhang L, Feng YH, Wu X. High glucose instigates tubulointerstitial injury by stimulating hetero-dimerization of adiponectin and angiotensin II receptors. Biochem Biophys Res Commun 2017;493:840-6. [PMID: 28870804 DOI: 10.1016/j.bbrc.2017.08.047] [Cited by in Crossref: 11] [Cited by in F6Publishing: 9] [Article Influence: 2.2] [Reference Citation Analysis]
Number Citing Articles
1 Hu M, Yang J, Xu Y, Liu J, Tang M. MDH1 and MDH2 Promote Cell Viability of Primary AT2 Cells by Increasing Glucose Uptake. Computational and Mathematical Methods in Medicine 2022;2022:1-15. [DOI: 10.1155/2022/2023500] [Reference Citation Analysis]
2 Haruhara K, Suzuki T, Wakui H, Azushima K, Kurotaki D, Kawase W, Uneda K, Kobayashi R, Ohki K, Kinguchi S, Yamaji T, Kato I, Ohashi K, Yamashita A, Tamura T, Tsuboi N, Yokoo T, Tamura K. Deficiency of the kidney tubular angiotensin II type1 receptor-associated protein ATRAP exacerbates streptozotocin-induced diabetic glomerular injury via reducing protective macrophage polarization. Kidney Int 2022:S0085-2538(22)00161-2. [PMID: 35240129 DOI: 10.1016/j.kint.2022.01.031] [Cited by in Crossref: 2] [Cited by in F6Publishing: 1] [Article Influence: 2.0] [Reference Citation Analysis]
3 Murayama MA, Chi HH, Matsuoka M, Ono T, Iwakura Y. The CTRP3-AdipoR2 Axis Regulates the Development of Experimental Autoimmune Encephalomyelitis by Suppressing Th17 Cell Differentiation. Front Immunol 2021;12:607346. [PMID: 34925309 DOI: 10.3389/fimmu.2021.607346] [Reference Citation Analysis]
4 Duan S, Lu F, Song D, Zhang C, Zhang B, Xing C, Yuan Y. Current Challenges and Future Perspectives of Renal Tubular Dysfunction in Diabetic Kidney Disease. Front Endocrinol (Lausanne) 2021;12:661185. [PMID: 34177803 DOI: 10.3389/fendo.2021.661185] [Reference Citation Analysis]
5 Bai J, Ma Q, Lan Y, Chen Y, Ma S, Li J, Liu C, Fu Z, Lu X, Huang Y, Li Y. Mitochondrial tRNA Mutation and Regulation of the Adiponectin Pathway in Maternally Inherited Hypertension in Chinese Han. Front Cell Dev Biol 2020;8:623450. [PMID: 33553162 DOI: 10.3389/fcell.2020.623450] [Reference Citation Analysis]
6 Liu Q, Lv S, Liu J, Liu S, Wang Y, Liu G. Mesenchymal stem cells modified with angiotensin-converting enzyme 2 are superior for amelioration of glomerular fibrosis in diabetic nephropathy. Diabetes Res Clin Pract 2020;162:108093. [PMID: 32109518 DOI: 10.1016/j.diabres.2020.108093] [Cited by in Crossref: 7] [Cited by in F6Publishing: 7] [Article Influence: 3.5] [Reference Citation Analysis]
7 Turu G, Balla A, Hunyady L. The Role of β-Arrestin Proteins in Organization of Signaling and Regulation of the AT1 Angiotensin Receptor. Front Endocrinol (Lausanne) 2019;10:519. [PMID: 31447777 DOI: 10.3389/fendo.2019.00519] [Cited by in Crossref: 15] [Cited by in F6Publishing: 13] [Article Influence: 5.0] [Reference Citation Analysis]
8 Wei PZ, Szeto CC. Mitochondrial dysfunction in diabetic kidney disease. Clin Chim Acta 2019;496:108-16. [PMID: 31276635 DOI: 10.1016/j.cca.2019.07.005] [Cited by in Crossref: 38] [Cited by in F6Publishing: 37] [Article Influence: 12.7] [Reference Citation Analysis]
9 Zha D, Yao T, Bao L, Gao P, Wu X. Telmisartan attenuates diabetic nephropathy progression by inhibiting the dimerization of angiotensin type-1 receptor and adiponectin receptor-1. Life Sciences 2019;221:109-20. [DOI: 10.1016/j.lfs.2019.01.044] [Cited by in Crossref: 3] [Cited by in F6Publishing: 4] [Article Influence: 1.0] [Reference Citation Analysis]
10 Wei Z, Kwan BC, Chow KM, Cheng PM, Luk CC, Lai KB, Li PK, Szeto CC. Urinary mitochondrial DNA level as a biomarker of tissue injury in non-diabetic chronic kidney diseases. BMC Nephrol 2018;19:367. [PMID: 30567508 DOI: 10.1186/s12882-018-1178-9] [Cited by in Crossref: 6] [Cited by in F6Publishing: 8] [Article Influence: 1.5] [Reference Citation Analysis]
11 Tóth AD, Turu G, Hunyady L, Balla A. Novel mechanisms of G-protein-coupled receptors functions: AT1 angiotensin receptor acts as a signaling hub and focal point of receptor cross-talk. Best Pract Res Clin Endocrinol Metab 2018;32:69-82. [PMID: 29678287 DOI: 10.1016/j.beem.2018.02.003] [Cited by in Crossref: 22] [Cited by in F6Publishing: 19] [Article Influence: 5.5] [Reference Citation Analysis]