Hepatitis B virus-persistent infection and innate immunity defect: Cell-related or virus-related?

doi:10.12998/wjcc.v6.i9.233

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World Journal of Clinical Cases

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2307-8960

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Clin Cases. Sep 6, 2018; 6(9): 233-241
Published online Sep 6, 2018. doi: 10.12998/wjcc.v6.i9.233

Table 1 Cytokines and factors implicated in causing inflammation in atherosclerosis

Cytokines/factors	Abbreviations
Hepatitis B virus	HBV
Chronic hepatitis B	CHB
Interferon	IFN
Pattern recognition receptors	PRRs
Toll-like receptors	TLRs
Melanoma differentiation-associated gene 5	MAD5
Retinoic acid-inducible gene I	RIG I
Stimulator of IFN genes	STING
Cyclic GMP-AMP synthase	cGAS
Human sodium taurocholate cotransporting polypeptide	hNTCP
Natural killer cell	NK cell
Tumor necrosis factor	TNF
Interleukin	IL
Kupffer cells	KCs
Dendritic cells	DCs
Plasmacytoid dendritic cells	pDC
Myeloid dendritic cells	mDC
Mitochondria-antiviral signaling protein	MAVS
Virus-induced signaling adaptor	VISA
Nucleoside analogs	NA

Citation: Tang J, Wu ZY, Dai RJ, Ma J, Gong GZ. Hepatitis B virus-persistent infection and innate immunity defect: Cell-related or virus-related? World J Clin Cases 2018; 6(9): 233-241
URL: https://www.wjgnet.com/2307-8960/full/v6/i9/233.htm
DOI: https://dx.doi.org/10.12998/wjcc.v6.i9.233