Total adenosine deaminase cases as an inflammatory biomarker of pleural effusion syndrome

Table 1 Demographic characteristics and causes of inflammatory and noninflammatory diseases in 157 patients with pleural effusion syndrome in the State of Rio de Janeiro, Brazil, from March 2015 to December 2019, n (%)/median (25^th-75^th percentiles)

Cause	Patient (n)	Prevalence (%)	Age	Female	Male
Non-inflammatory1	33	21.0	76.0 (63.0-86.25)	17.0 (52.0)	16.0 (48.0)
Inflammatory	124	79.0	58.0 (41.5-73.5)	66.0 (53.0)	58.0 (47.0)
Tuberculosis	44	28.0	39.0 (29.7-58.2)	22.0 (50.0)	22.0 (50.0)
Adenoc.	37	24.0	61.0 (45.0-77.0)	25 (68.0)	12 (32.0)
Simple PPE	15	10.0	67.0 (56.0-85.0)	6 (40.0)	9 (60.0)
CPPE/Empiema	8	5.0	52.5 (33.5-78.75)	2 (25.0)	6 (75.0)
Lymphoma	7	4.0	53.0 (47.0-63.0)	0 (0.0)	7 (100.0).
Squamous cell	7	4.0	66.0 (55.0-66.0)	4 (57.0)	3 (43.0)
Other2	6	4.0	73.0 (53.0-79.0)	4 (67.0)	2 (33.0)
Total	157	100.0	58.0 (41.75-73.25)	80 (51.0)	77 (49.0)

¹Transudates: Congestive heart failure (n = 26), chronic renal failure (n = 3), cirrhosis of liver with ascites (n = 3), and serum low total protein levels (n = 1).

²Other exudates: Pseudo-Meigs syndrome (n = 1), Dressler's syndrome (n = 3), chylothorax (n = 1), leukemia (n = 1).

CPPE: Complicated parapneumonic effusions.