Kidney injury in COVID-19

doi:10.5527/wjn.v9.i2.18

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Nov 29, 2020 (publication date) through Nov 4, 2024

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World Journal of Nephrology

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2220-6124

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Nephrol. Nov 29, 2020; 9(2): 18-32
Published online Nov 29, 2020. doi: 10.5527/wjn.v9.i2.18

Table 2 Summary of the mechanism of kidney injury in coronavirus disease 2019

Mechanism of kidney injury	Hypovolaemia	ARDS related AKI	Cytokine storm syndrome associated AKI	Direct viral invasion
Pre-renal	Fever causing insensible losses; Gastrointestinal volume losses	Haemodynamic instability; High positive end expiratory pressure /intrathoracic pressure; Right heart failure	Haemodynamic instability
Renal¹		Inflammation; Hypoxia/hypercapnia; Acid-base dysregulation; Tubular injury	Inflammation; Possible glomerulopathy and TMA ( hypercoagulability)	Inflammation; Possible Tubulopathy; Podocytopathy; Interstitial inflammation
Post renal

¹The most common intrinsic renal lesion observed is acute tubular necrosis. AKI: Acute kidney injury; ARDS: Acute respiratory distress syndrome; TMA: Thrombotic microangiopathy.

Citation: Ahmed AR, Ebad CA, Stoneman S, Satti MM, Conlon PJ. Kidney injury in COVID-19. World J Nephrol 2020; 9(2): 18-32
URL: https://www.wjgnet.com/2220-6124/full/v9/i2/18.htm
DOI: https://dx.doi.org/10.5527/wjn.v9.i2.18