Renal dopaminergic system: Pathophysiological implications and clinical perspectives

doi:10.5527/wjn.v4.i2.196

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May 6, 2015 (publication date) through Jan 3, 2025

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World Journal of Nephrology

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2220-6124

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Nephrol. May 6, 2015; 4(2): 196-212
Published online May 6, 2015. doi: 10.5527/wjn.v4.i2.196

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Figure 1 Dopamine receptors and regulation of redox state. Full line: Stimulation; Dotted line: Inhibition. D1R: Dopamine receptor subtype 1; D2R: Dopamine receptor subtype 2; D5R: Dopamine receptor subtype 5; NADPH: Nicotinamide adenine dinucleotide reduced form; SOD: Superoxide dismutase; HO-1: Heme oxygenase 1; HO-2: Heme oxygenase 2; PON2: Paraoxonase 2; DJ-1: Parkinson protein 7; GP: Glutathione peroxidase; GCT: Glutamyl cysteine transferase.

Citation: Choi MR, Kouyoumdzian NM, Rukavina Mikusic NL, Kravetz MC, Rosón MI, Rodríguez Fermepin M, Fernández BE. Renal dopaminergic system: Pathophysiological implications and clinical perspectives. World J Nephrol 2015; 4(2): 196-212
URL: https://www.wjgnet.com/2220-6124/full/v4/i2/196.htm
DOI: https://dx.doi.org/10.5527/wjn.v4.i2.196