Aging and uremia: Is there cellular and molecular crossover?

doi:10.5527/wjn.v4.i1.19

Advanced Search

BPG is committed to discovery and dissemination of knowledge

Home / Archive / Volume 4, Issue 1

This Article

Academic Content and Language Evaluation of This Article

Citation of this article

Corresponding Author of This Article

Research Domain of This Article

Article-Type of This Article

Open-Access Policy of This Article

Times Cited Counts in Google of This Article

Number of Hits and Downloads for This Article

Total Article Views (9533)

All Articles published online

The chart showing PDF series, WORD series, HTML series, Tables (1-1) series.

Item

Count

PDF

666

WORD

238

HTML

4015

Tables (1-1)

231

Sum=5150

Featured Article

The chart showing Browse series, Download series.

Item

Count

Browse

571

Download

1162

Sum=1733

Publishing Process of This Article

Item

Count

Browse

1234

Download

1416

Sum=2650

Feb 6, 2015 (publication date) through Dec 23, 2024

Times Cited of This Article

Times Cited (35)

Journal Information of This Article

Publication Name

World Journal of Nephrology

ISSN

2220-6124

Publisher of This Article

Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Review

World J Nephrol. Feb 6, 2015; 4(1): 19-30
Published online Feb 6, 2015. doi: 10.5527/wjn.v4.i1.19

Table 1 Events common to aging and uremia covered by this review

Aging	Uremia
TGF-β↑	TGF-β↑
Autophagy ↓	Autophagy ↓
Apoptosis ↑ (muscle)	Apoptosis ↑
Senescence ↑	Senescence ↑
Telomere shortening ↑	Telomere shortening ↑
Stem cell exhaustion ↑	Stem cell exhaustion ↑
Klotho ↓	Klotho ↓
AGEs ↑	AGEs ↑
Mitochondrial dysfunction ↑	Mitochondrial dysfunction ↑

TGF-β: Transforming growth factor beta; AGEs: Advanced glycation end products.

Citation: White WE, Yaqoob MM, Harwood SM. Aging and uremia: Is there cellular and molecular crossover? World J Nephrol 2015; 4(1): 19-30
URL: https://www.wjgnet.com/2220-6124/full/v4/i1/19.htm
DOI: https://dx.doi.org/10.5527/wjn.v4.i1.19